| Year |
Citation |
Score |
| 2023 |
Ashok D, Papanicolaou K, Sidor A, Wang M, Solhjoo S, Liu T, O'Rourke B. Mitochondrial Membrane Potential Instability on Reperfusion After Ischemia Does Not Depend on Mitochondrial Ca Uptake. The Journal of Biological Chemistry. 104708. PMID 37061004 DOI: 10.1016/j.jbc.2023.104708 |
0.349 |
|
| 2021 |
Yang N, Parker L, Yu J, Jones JW, Liu T, Papanicolaou KN, Talbot CC, Margulies KB, O'Rourke B, Kane M, Foster DB. Cardiac retinoic acid levels decline in heart failure. Jci Insight. PMID 33724958 DOI: 10.1172/jci.insight.137593 |
0.364 |
|
| 2020 |
Papanicolaou KN, Ashok D, Liu T, Bauer TM, Sun J, Li Z, da Costa E, D'Orleans CC, Nathan S, Lefer DJ, Murphy E, Paolocci N, Foster DB, O'Rourke B. Global knockout of ROMK potassium channel worsens cardiac ischemia-reperfusion injury but cardiomyocyte-specific knockout does not: Implications for the identity of mitoKATP. Journal of Molecular and Cellular Cardiology. PMID 32004507 DOI: 10.1016/J.Yjmcc.2020.01.010 |
0.414 |
|
| 2020 |
Ashok D, Papanicolaou K, Liu T, O'Rourke B. Reverse-Mode Mitochondrial Na+/Ca2+ Exchange, Not the MCU, is the Primary Mode of Ca2+ Import into the Mitochondria during Ischemia/Reperfusion in Neonatal Cardiac Myocytes Biophysical Journal. 118: 407a. DOI: 10.1016/J.Bpj.2019.11.2305 |
0.379 |
|
| 2019 |
Ashok D, Papanicolaou K, O′Rourke B. Mitochondrial Membrane Potential Oscillations Persist during Reperfusion After Ischemia in MCU Knockout Cardiomyocytes Biophysical Journal. 116: 269a-270a. DOI: 10.1016/J.Bpj.2018.11.1460 |
0.44 |
|
| 2016 |
Foster DB, Liu T, Kammers K, O'Meally RN, Yang N, Papanicolaou KN, Talbot C, Cole RN, O'Rourke B. Integrated Omic Analysis of a Guinea Pig Model of Heart Failure and Sudden Cardiac Death. Journal of Proteome Research. PMID 27399916 DOI: 10.1021/acs.jproteome.6b00149 |
0.373 |
|
| 2016 |
Maruyama S, Nakamura K, Papanicolaou KN, Sano S, Shimizu I, Asaumi Y, van den Hoff MJ, Ouchi N, Recchia FA, Walsh K. Follistatin-like 1 promotes cardiac fibroblast activation and protects the heart from rupture. Embo Molecular Medicine. PMID 27234440 DOI: 10.15252/Emmm.201506151 |
0.528 |
|
| 2014 |
Papanicolaou KN, O'Rourke B, Foster DB. Metabolism leaves its mark on the powerhouse: recent progress in post-translational modifications of lysine in mitochondria. Frontiers in Physiology. 5: 301. PMID 25228883 DOI: 10.3389/fphys.2014.00301 |
0.336 |
|
| 2014 |
Shimizu I, Aprahamian T, Kikuchi R, Shimizu A, Papanicolaou KN, MacLauchlan S, Maruyama S, Walsh K. Vascular rarefaction mediates whitening of brown fat in obesity. The Journal of Clinical Investigation. 124: 2099-112. PMID 24713652 DOI: 10.1172/Jci71643 |
0.413 |
|
| 2012 |
Papanicolaou KN, Kikuchi R, Ngoh GA, Coughlan KA, Dominguez I, Stanley WC, Walsh K. Mitofusins 1 and 2 are essential for postnatal metabolic remodeling in heart. Circulation Research. 111: 1012-26. PMID 22904094 DOI: 10.1161/Circresaha.112.274142 |
0.544 |
|
| 2012 |
Papanicolaou KN, Phillippo MM, Walsh K. Mitofusins and the mitochondrial permeability transition: the potential downside of mitochondrial fusion. American Journal of Physiology. Heart and Circulatory Physiology. 303: H243-55. PMID 22636681 DOI: 10.1152/Ajpheart.00185.2012 |
0.497 |
|
| 2012 |
Ngoh GA, Papanicolaou KN, Walsh K. Loss of mitofusin 2 promotes endoplasmic reticulum stress. The Journal of Biological Chemistry. 287: 20321-32. PMID 22511781 DOI: 10.1074/Jbc.M112.359174 |
0.438 |
|
| 2012 |
Papanicolaou KN, Ngoh GA, Dabkowski ER, O'Connell KA, Ribeiro RF, Stanley WC, Walsh K. Cardiomyocyte deletion of mitofusin-1 leads to mitochondrial fragmentation and improves tolerance to ROS-induced mitochondrial dysfunction and cell death. American Journal of Physiology. Heart and Circulatory Physiology. 302: H167-79. PMID 22037195 DOI: 10.1152/Ajpheart.00833.2011 |
0.563 |
|
| 2011 |
Papanicolaou KN, Khairallah RJ, Ngoh GA, Chikando A, Luptak I, O'Shea KM, Riley DD, Lugus JJ, Colucci WS, Lederer WJ, Stanley WC, Walsh K. Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes. Molecular and Cellular Biology. 31: 1309-28. PMID 21245373 DOI: 10.1128/Mcb.00911-10 |
0.582 |
|
| 2010 |
Papanicolaou KN, Streicher JM, Ishikawa TO, Herschman H, Wang Y, Walsh K. Preserved heart function and maintained response to cardiac stresses in a genetic model of cardiomyocyte-targeted deficiency of cyclooxygenase-2. Journal of Molecular and Cellular Cardiology. 49: 196-209. PMID 20399788 DOI: 10.1016/J.Yjmcc.2010.04.002 |
0.533 |
|
| 2009 |
Oshima Y, Ouchi N, Shimano M, Pimentel DR, Papanicolaou KN, Panse KD, Tsuchida K, Lara-Pezzi E, Lee SJ, Walsh K. Activin A and follistatin-like 3 determine the susceptibility of heart to ischemic injury. Circulation. 120: 1606-15. PMID 19805648 DOI: 10.1161/Circulationaha.109.872200 |
0.471 |
|
| 2008 |
Papanicolaou KN, Izumiya Y, Walsh K. Forkhead transcription factors and cardiovascular biology. Circulation Research. 102: 16-31. PMID 18174472 DOI: 10.1161/Circresaha.107.164186 |
0.41 |
|
| 2007 |
Shibata R, Izumiya Y, Sato K, Papanicolaou K, Kihara S, Colucci WS, Sam F, Ouchi N, Walsh K. Adiponectin protects against the development of systolic dysfunction following myocardial infarction. Journal of Molecular and Cellular Cardiology. 42: 1065-74. PMID 17499764 DOI: 10.1016/J.Yjmcc.2007.03.808 |
0.531 |
|
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