| Year |
Citation |
Score |
| 2024 |
Prem S, Dev B, Peng C, Mehta M, Alibutud R, Connacher RJ, St Thomas M, Zhou X, Matteson P, Xing J, Millonig JH, DiCicco-Bloom E. Dysregulation of mTOR signaling mediates common neurite and migration defects in both idiopathic and 16p11.2 deletion autism neural precursor cells. Elife. 13. PMID 38525876 DOI: 10.7554/eLife.82809 |
0.759 |
|
| 2022 |
Connacher R, Williams M, Prem S, Yeung PL, Matteson P, Mehta M, Markov A, Peng C, Zhou X, McDermott CR, Pang ZP, Flax J, Brzustowicz L, Lu CW, Millonig JH, et al. Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17: 1786. PMID 35830838 DOI: 10.1016/j.stemcr.2022.06.007 |
0.639 |
|
| 2022 |
Connacher R, Williams M, Prem S, Yeung PL, Matteson P, Mehta M, Markov A, Peng C, Zhou X, McDermott CR, Pang ZP, Flax J, Brzustowicz L, Lu CW, Millonig JH, et al. Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. PMID 35623351 DOI: 10.1016/j.stemcr.2022.04.019 |
0.659 |
|
| 2020 |
Turkalj L, Mehta M, Matteson P, Prem S, Williams M, Connacher RJ, DiCicco-Bloom E, Millonig JH. Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in Neurobiology. 25: 79-107. PMID 32578145 DOI: 10.1007/978-3-030-45493-7_4 |
0.625 |
|
| 2018 |
Connacher RJ, DiCicco-Bloom E, Millonig JH. Using Human Induced Neural Precursor Cells to Define Early Neurodevelopmental Defects in Syndromic and Idiopathic Autism Current Pharmacology Reports. 4: 422-435. DOI: 10.1007/S40495-018-0155-0 |
0.754 |
|
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