| Year |
Citation |
Score |
| 2023 |
Ma Y, Bendl J, Hartley BJ, Fullard JF, Abdelaal R, Ho SM, Kosoy R, Gochman P, Rapoport J, Hoffman GE, Brennand KJ, Roussos P. Activity-Dependent Transcriptional Program in NGN2+ Neurons Enriched for Genetic Risk for Brain-Related Disorders. Biological Psychiatry. PMID 37454787 DOI: 10.1016/j.biopsych.2023.07.003 |
0.375 |
|
| 2021 |
Powell SK, O'Shea C, Townsley K, Prytkova I, Dobrindt K, Elahi R, Iskhakova M, Lambert T, Valada A, Liao W, Ho SM, Slesinger PA, Huckins LM, Akbarian S, Brennand KJ. Induction of dopaminergic neurons for neuronal subtype-specific modeling of psychiatric disease risk. Molecular Psychiatry. PMID 34493831 DOI: 10.1038/s41380-021-01273-0 |
0.509 |
|
| 2021 |
Li A, Cartwright S, Yu A, Ho SM, Schrode N, Deans PJM, Matos MR, Garcia MF, Townsley KG, Zhang B, Brennand KJ. Using the dCas9-KRAB system to repress gene expression in hiPSC-derived neurons. Star Protocols. 2: 100580. PMID 34151300 DOI: 10.1016/j.xpro.2021.100580 |
0.757 |
|
| 2020 |
Wang M, Li A, Sekiya M, Beckmann ND, Quan X, Schrode N, Fernando MB, Yu A, Zhu L, Cao J, Lyu L, Horgusluoglu E, Wang Q, Guo L, Wang YS, ... ... Ho SM, et al. Transformative Network Modeling of Multi-omics Data Reveals Detailed Circuits, Key Regulators, and Potential Therapeutics for Alzheimer's Disease. Neuron. PMID 33238137 DOI: 10.1016/j.neuron.2020.11.002 |
0.739 |
|
| 2020 |
Matos MR, Ho SM, Schrode N, Brennand KJ. Integration of CRISPR-engineering and hiPSC-based models of psychiatric genomics. Molecular and Cellular Neurosciences. 103532. PMID 32712198 DOI: 10.1016/J.Mcn.2020.103532 |
0.786 |
|
| 2020 |
Barretto N, Zhang H, Powell SK, Fernando MB, Zhang S, Flaherty EK, Ho SM, Slesinger PA, Duan J, Brennand KJ. ASCL1- and DLX2-induced GABAergic neurons from hiPSC-derived NPCs. Journal of Neuroscience Methods. 334: 108548. PMID 32065989 DOI: 10.1016/J.Jneumeth.2019.108548 |
0.753 |
|
| 2019 |
Schrode N, Ho SM, Yamamuro K, Dobbyn A, Huckins L, Matos MR, Cheng E, Deans PJM, Flaherty E, Barretto N, Topol A, Alganem K, Abadali S, Gregory J, Hoelzli E, et al. Synergistic effects of common schizophrenia risk variants. Nature Genetics. PMID 31548722 DOI: 10.1038/S41588-019-0497-5 |
0.767 |
|
| 2019 |
Brennand K, Hoffman G, Schrode N, Flaherty E, Ho S. MODELING THE IMPACT OF RARE AND COMMON VARIANTS ON PREDISPOSITION TO SCHIZOPHRENIA USING STEM CELLS European Neuropsychopharmacology. 29: S1061. DOI: 10.1016/J.Euroneuro.2018.07.083 |
0.76 |
|
| 2018 |
Rajarajan P, Borrman T, Liao W, Schrode N, Flaherty E, Casiño C, Powell S, Yashaswini C, LaMarca EA, Kassim B, Javidfar B, Espeso-Gil S, Li A, Won H, Geschwind DH, ... Ho SM, et al. Neuron-specific signatures in the chromosomal connectome associated with schizophrenia risk. Science (New York, N.Y.). 362. PMID 30545851 DOI: 10.1126/Science.Aat4311 |
0.714 |
|
| 2017 |
Ho SM, Hartley BJ, Flaherty E, Rajarajan P, Abdelaal R, Obiorah I, Barretto N, Muhammad H, Phatnani HP, Akbarian S, Brennand KJ. Evaluating Synthetic Activation and Repression of Neuropsychiatric-Related Genes in hiPSC-Derived NPCs, Neurons, and Astrocytes. Stem Cell Reports. PMID 28757163 DOI: 10.1016/J.Stemcr.2017.06.012 |
0.766 |
|
| 2017 |
Jiang Y, Loh YE, Rajarajan P, Hirayama T, Liao W, Kassim BS, Javidfar B, Hartley BJ, Kleofas L, Park RB, Labonte B, Ho SM, Chandrasekaran S, Do C, Ramirez BR, et al. The methyltransferase SETDB1 regulates a large neuron-specific topological chromatin domain. Nature Genetics. PMID 28671686 DOI: 10.1038/Ng.3906 |
0.791 |
|
| 2016 |
Xu J, Hartley BJ, Kurup P, Phillips A, Topol A, Xu M, Ononenyi C, Foscue E, Ho SM, Baguley TD, Carty N, Barros CS, Müller U, Gupta S, Gochman P, et al. Inhibition of STEP61 ameliorates deficits in mouse and hiPSC-based schizophrenia models. Molecular Psychiatry. PMID 27752082 DOI: 10.1038/mp.2016.163 |
0.633 |
|
| 2015 |
Lee IS, Carvalho CM, Douvaras P, Ho SM, Hartley BJ, Zuccherato LW, Ladran IG, Siegel AJ, McCarthy S, Malhotra D, Sebat J, Rapoport J, Fossati V, Lupski JR, Levy DL, et al. Characterization of molecular and cellular phenotypes associated with a heterozygous CNTNAP2 deletion using patient-derived hiPSC neural cells. Npj Schizophrenia. 1. PMID 26985448 |
0.684 |
|
| 2015 |
Ho SM, Hartley BJ, Tcw J, Beaumont M, Stafford K, Slesinger PA, Brennand KJ. Rapid Ngn2-induction of Excitatory Neurons from hiPSC-Derived Neural Progenitor Cells. Methods (San Diego, Calif.). PMID 26626326 DOI: 10.1016/J.Ymeth.2015.11.019 |
0.723 |
|
| 2015 |
Ho SM, Topol A, Brennand KJ. From "directed differentiation" to "neuronal induction": modeling neuropsychiatric disease. Biomarker Insights. 10: 31-41. PMID 26045654 DOI: 10.4137/Bmi.S20066 |
0.715 |
|
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