Akira Inagaki
Affiliations: | 2009-2012 | Molecular Physiology and Biophysics | University of Iowa, Iowa City, IA |
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| Minakata T, Inagaki A, Yamamura A, et al. (2019) Calcium-Sensing Receptor Is Functionally Expressed in the Cochlear Perilymphatic Compartment and Essential for Hearing. Frontiers in Molecular Neuroscience. 12: 175 |
| Inagaki A, Frank CA, Usachev YM, et al. (2014) Pharmacological correction of gating defects in the voltage-gated Ca(v)2.1 Ca²⺠channel due to a familial hemiplegic migraine mutation. Neuron. 81: 91-102 |
| Inagaki A, Lee A. (2013) Developmental alterations in the biophysical properties of Ca(v) 1.3 Ca(2+) channels in mouse inner hair cells. Channels (Austin, Tex.). 7: 171-81 |
| Schrauwen I, Helfmann S, Inagaki A, et al. (2012) A mutation in CABP2, expressed in cochlear hair cells, causes autosomal-recessive hearing impairment. American Journal of Human Genetics. 91: 636-45 |
| Inagaki A, Ugawa S, Yamamura H, et al. (2008) The CaV3.1 T-type Ca2+channel contributes to voltage-dependent calcium currents in rat outer hair cells. Brain Research. 1201: 68-77 |
| Ugawa S, Inagaki A, Yamamura H, et al. (2006) Acid-sensing ion channel-1b in the stereocilia of mammalian cochlear hair cells. Neuroreport. 17: 1235-9 |