Akira Inagaki - Publications

Affiliations: 
2009-2012 Molecular Physiology and Biophysics University of Iowa, Iowa City, IA 

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Minakata T, Inagaki A, Yamamura A, Yamamura H, Sekiya S, Murakami S. Calcium-Sensing Receptor Is Functionally Expressed in the Cochlear Perilymphatic Compartment and Essential for Hearing. Frontiers in Molecular Neuroscience. 12: 175. PMID 31379498 DOI: 10.3389/fnmol.2019.00175  0.542
2014 Inagaki A, Frank CA, Usachev YM, Benveniste M, Lee A. Pharmacological correction of gating defects in the voltage-gated Ca(v)2.1 Ca²⁺ channel due to a familial hemiplegic migraine mutation. Neuron. 81: 91-102. PMID 24411734 DOI: 10.1016/J.Neuron.2013.10.056  0.53
2013 Inagaki A, Lee A. Developmental alterations in the biophysical properties of Ca(v) 1.3 Ca(2+) channels in mouse inner hair cells. Channels (Austin, Tex.). 7: 171-81. PMID 23510940 DOI: 10.4161/chan.24104  0.52
2012 Schrauwen I, Helfmann S, Inagaki A, Predoehl F, Tabatabaiefar MA, Picher MM, Sommen M, Seco CZ, Oostrik J, Kremer H, Dheedene A, Claes C, Fransen E, Chaleshtori MH, Coucke P, et al. A mutation in CABP2, expressed in cochlear hair cells, causes autosomal-recessive hearing impairment. American Journal of Human Genetics. 91: 636-45. PMID 22981119 DOI: 10.1016/J.Ajhg.2012.08.018  0.512
2008 Inagaki A, Ugawa S, Yamamura H, Murakami S, Shimada S. The CaV3.1 T-type Ca2+channel contributes to voltage-dependent calcium currents in rat outer hair cells. Brain Research. 1201: 68-77. PMID 18294617 DOI: 10.1016/J.Brainres.2008.01.058  0.585
2006 Ugawa S, Inagaki A, Yamamura H, Ueda T, Ishida Y, Kajita K, Shimizu H, Shimada S. Acid-sensing ion channel-1b in the stereocilia of mammalian cochlear hair cells. Neuroreport. 17: 1235-9. PMID 16951561 DOI: 10.1097/01.Wnr.0000233093.67289.66  0.301
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