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Erwan Bezard

Affiliations: 
Université de Bordeaux, Talence, Nouvelle-Aquitaine, France 
Area:
neurodegenerative disorders
Website:
http://www.inb.u-bordeaux2.fr/dev/FR/equipe.php?equipe=Physiopathologie%20des%20syndromes%20parkinsoniens
Google:
"Erwan Bezard"
Bio:

Bezard has authored or co-authored over 90 professional publications in the field of neurobiology, most of which are on Parkinson's disease and related disorders. He is perhaps best known for the reappraisal of both the identification of the nature of compensatory mechanisms that mask the progression Parkinson's disease and their sequential activation in non-human primate models that mimic Parkinson’s progression. His current research interests include the study of the compensatory mechanisms, the levodopa-induced dyskinesia, the basic pathophysiology of basal ganglia circuitry, and the development of new strategies to alleviate symptoms and/or to slow disease progression.

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Mean distance: 15.8 (cluster 6)
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Parents

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Bernard H. Bioulac grad student 1995-1998 Université de Bordeaux
Christian E. Gross grad student 1995-1998 Université de Bordeaux
 (PhD thesis)
Jonathan M. Brotchie post-doc 1999-2000 The University of Manchester
Alan R. Crossman post-doc 1999-2000 The University of Manchester

Children

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Sandra Dovero grad student Université de Bordeaux
Michel Engeln grad student 2010-2013 Université de Bordeaux
Mathieu Bourdenx grad student 2012-2015 Université de Bordeaux
Salvatore Novello grad student 2014-2019 Université de Bordeaux

Collaborators

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Publications

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Bendetowicz D, Planche V, Bezard E, et al. (2025) Biological definitions of synucleinopathies should be anchored in clinical trajectories and encompass the complex biology of the disease. Journal of Parkinson's Disease. 1877718X241313443
Katrib C, Hladky H, Timmerman K, et al. (2024) Magnetic resonance imaging characterization of an α-synuclein model of Parkinson's disease. The European Journal of Neuroscience
Sikora J, Dovero S, Kinet R, et al. (2024) Nigral ATP13A2 depletion induces Parkinson's disease-related neurodegeneration in a pilot study in non-human primates. Npj Parkinson's Disease. 10: 141
Bezard E, Teil M, Arotcarena ML, et al. (2024) Corrigendum: Modeling Parkinson's Disease in Primates. Cold Spring Harbor Perspectives in Medicine
Bezard E, Teil M, Arotcarena ML, et al. (2024) Modeling Parkinson's Disease in Primates. Cold Spring Harbor Perspectives in Medicine
Cirillo R, Duperrier S, Parekh P, et al. (2024) Striatal Serotonin 4 Receptor is Increased in Experimental Parkinsonism and Dyskinesia. Journal of Parkinson's Disease
Rocha E, Chamoli M, Chinta SJ, et al. (2023) Aging, Parkinson's Disease, and Models: What Are the Challenges? Aging Biology. 1
Darricau M, Dou C, Kinet R, et al. (2023) Tau seeds from Alzheimer's disease brains trigger tau spread in macaques while oligomeric-Aβ mediates pathology maturation. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
Teil M, Dovero S, Bourdenx M, et al. (2023) Cortical Lewy body injections induce long-distance pathogenic alterations in the non-human primate brain. Npj Parkinson's Disease. 9: 135
Serra M, Di Maio A, Bassareo V, et al. (2023) Perturbation of serine enantiomers homeostasis in the striatum of MPTP-lesioned monkeys and mice reflects the extent of dopaminergic midbrain degeneration. Neurobiology of Disease. 106226
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