Ken Nakamura

Affiliations: 
University of California, San Francisco, San Francisco, CA 
Area:
Mitochondrial Biology in Neurodegenerative Disease
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"Ken Nakamura"
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Publications

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Rademacher K, Doric Z, Haddad D, et al. (2024) Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration. Biorxiv : the Preprint Server For Biology
Rademacher K, Nakamura K. (2023) Role of dopamine neuron activity in Parkinson's disease pathophysiology. Experimental Neurology. 114645
Doric Z, Li H, Nakamura K. (2021) The PINK1 advantage: recycling mitochondria in times of trouble? Autophagy. 18: 231-232
Nguyen MK, McAvoy K, Liao SC, et al. (2021) Mouse midbrain dopaminergic neurons survive loss of the PD-associated mitochondrial protein CHCHD2. Human Molecular Genetics
Li H, Doric Z, Berthet A, et al. (2021) Longitudinal tracking of neuronal mitochondria delineates PINK1/Parkin-dependent mechanisms of mitochondrial recycling and degradation. Science Advances. 7
Zhang J, Shang Y, Kamiya S, et al. (2019) Loss of HIPK2 protects neurons from mitochondrial toxins by regulating Parkin protein turnover. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Mendelsohn BA, Bennett NK, Darch MA, et al. (2018) A high-throughput screen of real-time ATP levels in individual cells reveals mechanisms of energy failure. Plos Biology. 16: e2004624
Pathak D, Berthet A, Bendor JT, et al. (2017) Loss of α-Synuclein Does Not Affect Mitochondrial Bioenergetics in Rodent Neurons. Eneuro. 4
Orr AL, Rutaganira FU, de Roulet D, et al. (2017) Long-term oral kinetin does not protect against α-synuclein-induced neurodegeneration in rodent models of Parkinson's disease. Neurochemistry International
Haddad D, Nakamura K. (2017) PINK1-Based Screen Shines Light on Autophagy Enhancers for Parkinson's Disease. Cell Chemical Biology. 24: 429-430
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