Michelle D. Amaral
Affiliations: | University of Alabama, Birmingham, Birmingham, AL, United States |
Area:
Synapses, BDNF, HippocampusGoogle:
"Michelle Amaral"Mean distance: 14.93 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Lucas Pozzo-Miller | grad student | 2003-2008 | UAB | |
| (TRP-ing down a TRK: A new role for transient receptor potential channels as novel mediators of brain -derived neurotrophic factor actions at both sides of the excitatory synapse.) | ||||
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Publications
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| Baker K, Gordon SL, Melland H, et al. (2018) SYT1-associated neurodevelopmental disorder: a case series. Brain : a Journal of Neurology |
| Silva MC, Amaral MD, Morimoto RI. (2013) Neuronal reprograming of protein homeostasis by calcium-dependent regulation of the heat shock response. Plos Genetics. 9: e1003711 |
| Tian Y, Schreiber R, Wanitchakool P, et al. (2013) Control of TMEM16A by INO-4995 and other inositolphosphates. British Journal of Pharmacology. 168: 253-65 |
| Leuner K, Li W, Amaral MD, et al. (2013) Hyperforin modulates dendritic spine morphology in hippocampal pyramidal neurons by activating Ca(2+) -permeable TRPC6 channels. Hippocampus. 23: 40-52 |
| Amaral MD, Pozzo-Miller L. (2012) Intracellular Ca2+ stores and Ca2+ influx are both required for BDNF to rapidly increase quantal vesicular transmitter release. Neural Plasticity. 2012: 203536 |
| Martins JR, Kongsuphol P, Sammels E, et al. (2011) F508del-CFTR increases intracellular Ca(2+) signaling that causes enhanced calcium-dependent Cl(-) conductance in cystic fibrosis. Biochimica Et Biophysica Acta. 1812: 1385-92 |
| Li Y, Calfa G, Inoue T, et al. (2010) Activity-dependent release of endogenous BDNF from mossy fibers evokes a TRPC3 current and Ca2+ elevations in CA3 pyramidal neurons. Journal of Neurophysiology. 103: 2846-56 |
| Amaral MD, Pozzo-Miller L. (2009) The dynamics of excitatory synapse formation on dendritic spines. Cellscience. 5: 19-25 |
| Amaral MD, Pozzo-Miller L. (2007) BDNF induces calcium elevations associated with IBDNF, a nonselective cationic current mediated by TRPC channels. Journal of Neurophysiology. 98: 2476-82 |
| Amaral MD, Pozzo-Miller L. (2007) TRPC3 channels are necessary for brain-derived neurotrophic factor to activate a nonselective cationic current and to induce dendritic spine formation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 5179-89 |