Pavan K. Auluck, MD PhD
Affiliations: | Pathology | Massachusetts Institute of Technology and Harvard Medical School and Masssachusetts General Hospital, Boston, MA, United States |
Area:
models of neurodegenerative diseaseGoogle:
"Pavan Auluck"Bio:
http://web.wi.mit.edu/lindquist/pub/NeurodegenDisease.html
http://www2.massgeneral.org/pathology/train/sp-lit.htm
Mean distance: 14.73 (cluster 24) | S | N | B | C | P |
Parents
Sign in to add mentorNancy Mara Bonini | grad student | 1998-2003 | Penn | |
(Modelling Parkinson's disease in Drosophila: The protective role of molecular chaperones.) | ||||
Matthew P. Frosch | post-doc | 2004- | Massachusetts General Hospital & Harvard Medical School | |
Susan L. Lindquist | post-doc | 2008- | Whitehead Institute (FlyTree) | |
E. Tessa Hedley-Whyte | post-doc | 2006-2008 | Harvard Medical School and Masssachusetts General Hospital | |
David N. Louis | post-doc | 2006-2008 | Massachusetts General Hospital & Harvard Medical School |
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Publications
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Weihofen A, Liu Y, Arndt JW, et al. (2018) Development of an aggregate-selective, human-derived α-synuclein antibody BIIB054 that ameliorates disease phenotypes in Parkinson's disease models. Neurobiology of Disease |
Chung CY, Khurana V, Yi S, et al. (2017) In Situ Peroxidase Labeling and Mass-Spectrometry Connects Alpha-Synuclein Directly to Endocytic Trafficking and mRNA Metabolism in Neurons. Cell Systems |
Khurana V, Peng J, Chung CY, et al. (2017) Genome-Scale Networks Link Neurodegenerative Disease Genes to α-Synuclein through Specific Molecular Pathways. Cell Systems |
Caraveo G, Auluck PK, Whitesell L, et al. (2014) Calcineurin determines toxic versus beneficial responses to α-synuclein. Proceedings of the National Academy of Sciences of the United States of America. 111: E3544-52 |
Chung CY, Khurana V, Auluck PK, et al. (2013) Identification and rescue of α-synuclein toxicity in Parkinson patient-derived neurons. Science (New York, N.Y.). 342: 983-7 |
di Tomaso E, Snuderl M, Kamoun WS, et al. (2011) Glioblastoma recurrence after cediranib therapy in patients: lack of "rebound" revascularization as mode of escape. Cancer Research. 71: 19-28 |
Auluck PK, Caraveo G, Lindquist S. (2010) α-Synuclein: membrane interactions and toxicity in Parkinson's disease. Annual Review of Cell and Developmental Biology. 26: 211-33 |
Su LJ, Auluck PK, Outeiro TF, et al. (2010) Compounds from an unbiased chemical screen reverse both ER-to-Golgi trafficking defects and mitochondrial dysfunction in Parkinson's disease models. Disease Models & Mechanisms. 3: 194-208 |
Steele AD, Zhou Z, Jackson WS, et al. (2009) Context dependent neuroprotective properties of prion protein (PrP). Prion. 3: 240-9 |
Yeger-Lotem E, Riva L, Su LJ, et al. (2009) Bridging high-throughput genetic and transcriptional data reveals cellular responses to alpha-synuclein toxicity. Nature Genetics. 41: 316-23 |