Rudolph E. Tanzi, PhD, Harvard University

Affiliations: 
Neurology Harvard Medical School, Boston, MA, United States 
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=11
Website:
http://www.mghmind.org/Faculty/tanzi_rudy.html
Google:
"Rudolph Tanzi"
Bio:

http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=11

Mean distance: 14.78 (cluster 28)
 
Cross-listing: Alzheimer's Tree

BETA: Related publications

Publications

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Dujardin S, Commins C, Lathuiliere A, et al. (2020) Tau molecular diversity contributes to clinical heterogeneity in Alzheimer's disease. Nature Medicine
Minaeva O, Sarangi S, Ledoux DM, et al. (2020) In Vivo Quasi-Elastic Light Scattering Eye Scanner Detects Molecular Aging in Humans. The Journals of Gerontology. Series a, Biological Sciences and Medical Sciences
Cable J, Holtzman DM, Hyman BT, et al. (2020) Alternatives to amyloid for Alzheimer's disease therapies-a symposium report. Annals of the New York Academy of Sciences
Kwak SS, Washicosky KJ, Brand E, et al. (2020) Amyloid-β42/40 ratio drives tau pathology in 3D human neural cell culture models of Alzheimer's disease. Nature Communications. 11: 1377
Zhang X, Zhang C, Prokopenko D, et al. (2020) Negative Evidence for a Role of APH1B T27I Variant in Alzheimer's Disease. Human Molecular Genetics
Prikhodko O, Rynearson KD, Sekhon T, et al. (2020) The GSM BPN-15606 as a Potential Candidate for Preventative Therapy in Alzheimer's Disease. Journal of Alzheimer's Disease : Jad
Shin Y, Choi SH, Kim E, et al. (2019) Blood-Brain Barrier Dysfunction in a 3D In Vitro Model of Alzheimer's Disease. Advanced Science (Weinheim, Baden-Wurttemberg, Germany). 6: 1900962
Suh J, Romano DM, Nitschke L, et al. (2019) Loss of Ataxin-1 Potentiates Alzheimer's Pathogenesis by Elevating Cerebral BACE1 Transcription. Cell. 178: 1159-1175.e17
Kunkle BW, Grenier-Boley B, Sims R, et al. (2019) Author Correction: Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing. Nature Genetics
Moir RD, Tanzi RE. (2019) Low Evolutionary Selection Pressure in Senescence Does Not Explain the Persistence of Aβ in the Vertebrate Genome. Frontiers in Aging Neuroscience. 11: 70
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