William King Engel, MD
Affiliations: | Neurology | University of Southern California, Los Angeles, CA, United States |
Area:
neuromuscular diseasesWebsite:
http://www.uscneurology.org/schools/medicine/departments/neurology/nmuscular/people_details.asp?ID=13Google:
"William Engel"Mean distance: 106866
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Publications
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| Askanas V, Engel WK, Nogalska A. (2015) Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy. Biochimica Et Biophysica Acta. 1852: 633-43 |
| Cacciottolo M, Nogalska A, D'Agostino C, et al. (2013) Chaperone-mediated autophagy components are upregulated in sporadic inclusion-body myositis muscle fibres. Neuropathology and Applied Neurobiology. 39: 750-61 |
| D'Agostino C, Nogalska A, Cacciottolo M, et al. (2011) Abnormalities of NBR1, a novel autophagy-associated protein, in muscle fibers of sporadic inclusion-body myositis. Acta Neuropathologica. 122: 627-36 |
| Nogalska A, D'Agostino C, Engel WK, et al. (2011) Novel demonstration of conformationally modified tau in sporadic inclusion-body myositis muscle fibers. Neuroscience Letters. 503: 229-33 |
| D'Agostino C, Nogalska A, Engel WK, et al. (2011) In sporadic inclusion body myositis muscle fibres TDP-43-positive inclusions are less frequent and robust than p62 inclusions, and are not associated with paired helical filaments. Neuropathology and Applied Neurobiology. 37: 315-20 |
| Nogalska A, D'Agostino C, Engel WK, et al. (2010) Novel demonstration of amyloid-β oligomers in sporadic inclusion-body myositis muscle fibers. Acta Neuropathologica. 120: 661-6 |
| Yan J, Deng HX, Siddique N, et al. (2010) Frameshift and novel mutations in FUS in familial amyotrophic lateral sclerosis and ALS/dementia. Neurology. 75: 807-14 |
| Askanas V, Engel WK, Nogalska A. (2009) Inclusion body myositis: a degenerative muscle disease associated with intra-muscle fiber multi-protein aggregates, proteasome inhibition, endoplasmic reticulum stress and decreased lysosomal degradation. Brain Pathology (Zurich, Switzerland). 19: 493-506 |
| Wójcik S, Nogalska A, Engel WK, et al. (2008) Myostatin and its precursor protein are increased in the skeletal muscle of patients with Type-II muscle fibre atrophy. Folia Morphologica. 67: 6-12 |
| Wojcik S, Paciello O, Engel WK, et al. (2007) G.P.13.15 In sporadic inclusion-body myositis muscle-fiber (s-IBM) cytoplasm, cytochrome C aggregates with α-synuclein and amyloid-β precursor protein (AβPP), but does not activate caspase-3 Neuromuscular Disorders. 17: 853 |