Karen Gylys
Affiliations: | Neuroscience | University of California, Los Angeles, Los Angeles, CA |
Area:
Neurobiology BiologyGoogle:
"Karen Gylys"Mean distance: 53433
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Publications
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| Landry O, François A, Oye Mintsa Mi-Mba MF, et al. (2023) Postsynaptic protein Shank3a deficiency synergizes with Alzheimer's disease neuropathology to impair cognitive performance in the 3xTg-AD murine model. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Cohn W, Melnik M, Huang C, et al. (2021) Multi-Omics Analysis of Microglial Extracellular Vesicles From Human Alzheimer's Disease Brain Tissue Reveals Disease-Associated Signatures. Frontiers in Pharmacology. 12: 766082 |
| Miyoshi E, Bilousova T, Melnik M, et al. (2021) Exosomal tau with seeding activity is released from Alzheimer's disease synapses, and seeding potential is associated with amyloid beta. Laboratory Investigation; a Journal of Technical Methods and Pathology |
| Bilousova T, Simmons BJ, Knapp RR, et al. (2020) Dual Neutral Sphingomyelinase-2/Acetylcholinesterase Inhibitors for the treatment of Alzheimer's disease. Acs Chemical Biology |
| Bilousova T, Melnik M, Miyoshi E, et al. (2019) Apolipoprotein E/Aβ Complex Accumulates in AD Cortical Synapses via apoE Receptors and Is Enhanced by APOE4. The American Journal of Pathology |
| Ma QL, Teng E, Zuo X, et al. (2018) Neuronal pentraxin 1: A synaptic-derived plasma biomarker in Alzheimer's disease. Neurobiology of Disease |
| Abud EM, Ramirez RN, Martinez ES, et al. (2017) iPSC-Derived Human Microglia-like Cells to Study Neurological Diseases. Neuron. 94: 278-293.e9 |
| Gylys KH, Bilousova T. (2017) Flow Cytometry Analysis and Quantitative Characterization of Tau in Synaptosomes from Alzheimer's Disease Brains. Methods in Molecular Biology (Clifton, N.J.). 1523: 273-284 |
| Prieto GA, Trieu BH, Dang CT, et al. (2016) Pharmacological rescue of long-term potentiation in Alzheimer diseased synapses. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Bilousova T, Miller CA, Poon WW, et al. (2016) Synaptic Amyloid-β Oligomers Precede p-Tau and Differentiate High Pathology Control Cases. The American Journal of Pathology. 186: 185-98 |