Christian Haass, PhD, University of Heidelberg 1989
Affiliations: | Biochemistry | Ludwig-Maximilians-Universität München, München, Bayern, Germany |
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?n=&u_id=25Website:
http://www.cipsm.de/en/aboutCIPSM/members/haassChristian/index.html?style=0Google:
"Christian Haass"Bio:
http://en.wikipedia.org/wiki/Christian_Haass
Mean distance: 17.66 (cluster 31) | S | N | B | C | P |
Cross-listing: Alzheimer's Tree - Chemistry Tree
Parents
Sign in to add mentorDennis J. Selkoe | post-doc | 1990-1992 | Brigham and Women's Hospital, Harvard Medical School |
Children
Sign in to add traineeLaura T. Haas | research assistant | 2012-2012 | Universität München |
Ulf Dettmer | grad student | (Chemistry Tree) | |
Gil G. Westmeyer | grad student | (Chemistry Tree) | |
Xianyuan Xiang | grad student | 2013- | Universität München |
Stefan Prokop | grad student | 2002-2005 | |
Samira Parhizkar | grad student | 2014-2019 | |
Dieter Edbauer | post-doc | LMU |
Collaborators
Sign in to add collaboratorMatthias Brendel | collaborator | ||
Yuetiva Deming | collaborator | 2017- | Ludwig Maximilian University Munich |
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Shojaei M, Schaefer R, Schlepckow K, et al. (2024) PET imaging of microglia in Alzheimer's disease using copper-64 labeled TREM2 antibodies. Theranostics. 14: 6319-6336 |
Mori K, Arzberger T, Grässer FA, et al. (2024) Correction to: Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteins. Acta Neuropathologica. 148: 48 |
Zatcepin A, Gnörich J, Rauchmann BS, et al. (2024) Regional desynchronization of microglial activity is associated with cognitive decline in Alzheimer's disease. Molecular Neurodegeneration. 19: 64 |
Parhizkar S, Arzberger T, Brendel M, et al. (2024) Author Correction: Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoE. Nature Neuroscience |
Hu Y, Hruscha A, Pan C, et al. (2024) Mis-localization of endogenous TDP-43 leads to ALS-like early-stage metabolic dysfunction and progressive motor deficits. Molecular Neurodegeneration. 19: 50 |
Ramalingam N, Haass C, Dettmer U. (2024) Physiological roles of α-synuclein serine-129 phosphorylation - not an oxymoron. Trends in Neurosciences. 47: 480-490 |
Riemenschneider H, Guo Q, Bader J, et al. (2024) Author Correction: Gel-like inclusions of C-terminal fragments of TDP-43 sequester stalled proteasomes in neurons. Embo Reports |
Reich M, Simon MJ, Polke B, et al. (2024) Peripheral expression of brain-penetrant progranulin rescues pathologies in mouse models of frontotemporal lobar degeneration. Science Translational Medicine. 16: eadj7308 |
Palleis C, Franzmeier N, Weidinger E, et al. (2023) Association of Neurofilament Light Chain, [F]PI-2620 Tau-PET, TSPO-PET, and Clinical Progression in Patients With β-Amyloid-Negative CBS. Neurology. 102: e207901 |
Schlepckow K, Morenas-Rodríguez E, Hong S, et al. (2023) Stimulation of TREM2 with agonistic antibodies-an emerging therapeutic option for Alzheimer's disease. The Lancet. Neurology. 22: 1048-1060 |