Emily M. Rocha
Affiliations: | 2015- | University of Pittsburgh, Pittsburgh, PA, United States |
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| Sturchio A, Rocha EM, Kauffman MA, et al. (2024) Recalibrating the Why and Whom of Animal Models in Parkinson Disease: A Clinician's Perspective. Brain Sciences. 14 |
| Van Laar AD, Webb KR, Keeney MT, et al. (2023) Transient exposure to rotenone causes degeneration and progressive parkinsonian motor deficits, neuroinflammation, and synucleinopathy. Npj Parkinson's Disease. 9: 121 |
| Mamais A, Wallings R, Rocha EM. (2023) Disease mechanisms as subtypes: Lysosomal dysfunction in the endolysosomal Parkinson's disease subtype. Handbook of Clinical Neurology. 193: 33-51 |
| Rocha EM, Keeney MT, Di Maio R, et al. (2022) LRRK2 and idiopathic Parkinson's disease. Trends in Neurosciences |
| De Miranda BR, Castro SL, Rocha EM, et al. (2021) The industrial solvent trichloroethylene induces LRRK2 kinase activity and dopaminergic neurodegeneration in a rat model of Parkinson's disease. Neurobiology of Disease. 105312 |
| De Miranda BR, Rocha EM, Castro SL, et al. (2020) Protection from α-Synuclein induced dopaminergic neurodegeneration by overexpression of the mitochondrial import receptor TOM20. Npj Parkinson's Disease. 6: 38 |
| Rocha EM, De Miranda BR, Castro S, et al. (2019) LRRK2 inhibition prevents endolysosomal deficits seen in human Parkinson's disease. Neurobiology of Disease. 104626 |
| De Miranda BR, Fazzari M, Rocha EM, et al. (2019) Sex differences in rotenone sensitivity reflect the male-to-female ratio in human Parkinson's disease incidence. Toxicological Sciences : An Official Journal of the Society of Toxicology |
| Di Maio R, Hoffman EK, Rocha EM, et al. (2018) LRRK2 activation in idiopathic Parkinson's disease. Science Translational Medicine. 10 |
| Hallett PJ, Huebecker M, Brekk OR, et al. (2018) Glycosphingolipid levels and glucocerebrosidase activity are altered in normal aging of the mouse brain. Neurobiology of Aging. 67: 189-200 |