Audrey Claing, Ph.D

Affiliations: 
Pharmacologie Université de Montréal, Montréal, Canada 
Area:
GPCRs
Website:
http://www.pharmacologie.umontreal.ca/professeurs_chercheurs/claing/index.html
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"Audrey Claing"
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Publications

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Giubilaro J, Schuetz DA, Stepniewski TM, et al. (2021) Discovery of a dual Ras and ARF6 inhibitor from a GPCR endocytosis screen. Nature Communications. 12: 4688
Charles R, Bourmoum M, Campbell S, et al. (2019) Methods to Investigate the β-Arrestin-Mediated Control of ARF6 Activation to Regulate Trafficking and Actin Cytoskeleton Remodeling. Methods in Molecular Biology (Clifton, N.J.). 1957: 159-168
Charles R, Bourmoum M, Claing A. (2018) ARF GTPases control phenotypic switching of vascular smooth muscle cells through the regulation of actin function and actin dependent gene expression. Cellular Signalling
Aubé A, Campbell S, Schmitzer AR, et al. (2017) Ultra-low fouling methylimidazolium modified surfaces for the detection of HER2 in breast cancer cell lysates. The Analyst
Bourmoum M, Claing A. (2017) The GTPase ARF6 Regulates Mitotic Exit and Cell Cycle Progression Journal of Pharmacological and Toxicological Methods. 88: 169
Chingle R, Ratni S, Claing A, et al. (2016) Application of constrained aza-valine analogs for Smac mimicry. Biopolymers
Schlienger S, Campbell S, Pasquin S, et al. (2016) ADP-ribosylation factor 1 expression regulates epithelial-mesenchymal transition and predicts poor clinical outcome in triple-negative breast cancer. Oncotarget
Bourmoum M, Charles R, Claing A. (2016) The GTPase ARF6 Controls ROS Production to Mediate Angiotensin II-Induced Vascular Smooth Muscle Cell Proliferation. Plos One. 11: e0148097
Charles R, Namkung Y, Cotton M, et al. (2015) βarrestin-Mediated Angiotensin II Signaling Controls the Activation of ARF6 and Endocytosis in Migration of Vascular Smooth Muscle Cells. The Journal of Biological Chemistry
Haines E, Schlienger S, Claing A. (2015) The small GTPase ADP-Ribosylation Factor 1 mediates the sensitivity of triple negative breast cancer cells to EGFR tyrosine kinase inhibitors. Cancer Biology & Therapy. 0
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