Luciano D'Adamio
Affiliations: | Yeshiva University, New York, NY, United States |
Area:
Neuroscience Biology, Pathology, Molecular BiologyGoogle:
"Luciano D'Adamio"Mean distance: 21373.2
Children
Sign in to add traineeEnrico Ghersi | grad student | 2004 | Yeshiva University |
Meir H. Scheinfeld | grad student | 2005 | Yeshiva University |
Dawang Zhou | grad student | 2006 | Yeshiva University |
Robert Tamayev | grad student | 2013 | Yeshiva University |
Tao Yin | grad student | 2017-2022 | Rutgers - The State University of New Jersey |
Metin Yesiltepe | post-doc | 2021- | Rutgers New Jersey Medical School |
Tao Yin | post-doc | 2022- | Rutgers New Jersey Medical School |
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Yin T, Yesiltepe M, D'Adamio L. (2024) Functional BRI2-TREM2 interactions in microglia: implications for Alzheimer's and related dementias. Embo Reports |
D'Adamio L. (2023) Transfixed by transgenics: how pathology assumptions are slowing progress in Alzheimer's disease and related dementia research. Embo Molecular Medicine. 15: e18479 |
Yesiltepe M, Yin T, D'Adamio L. (2023) Beyond amyloid: altered gene function in neurodegenerative diseases. Aging. 15: 9235-9237 |
Yin T, D'Adamio L. (2023) BRI2-mediated regulation of TREM2 processing in microglia and its potential implications for Alzheimer's disease and related dementias. Biorxiv : the Preprint Server For Biology |
Puliatti G, Li Puma DD, Aceto G, et al. (2023) Intracellular accumulation of tau oligomers in astrocytes and their synaptotoxic action rely on Amyloid Precursor Protein Intracellular Domain-dependent expression of Glypican-4. Progress in Neurobiology. 227: 102482 |
Tambini MD, Yin T, Yesiltepe M, et al. (2023) Aβ43 levels determine the onset of pathological amyloid deposition. The Journal of Biological Chemistry. 104868 |
Yesiltepe M, Yin T, Tambini MD, et al. (2022) Late-long-term potentiation magnitude, but not Aβ levels and amyloid pathology, is associated with behavioral performance in a rat knock-in model of Alzheimer disease. Frontiers in Aging Neuroscience. 14: 1040576 |
Pham H, Yin T, D'Adamio L. (2022) Initial assessment of the spatial learning, reversal, and sequencing task capabilities of knock-in rats with humanizing mutations in the Aβ-coding region of App. Plos One. 17: e0263546 |
Yin T, Yao W, Norris KA, et al. (2021) A Familial Danish dementia rat shows impaired pre- and postsynaptic glutamatergic transmission. The Journal of Biological Chemistry. 101089 |
Tambini MD, D'Adamio L. (2020) Knock-in rats with homozygous PSEN1 Alzheimer mutation are viable and show selective γ-secretase activity loss causing low Aβ40/42 and high Aβ43. The Journal of Biological Chemistry. 295: 7442-7451 |