Cole M. Haynes, Ph.D.
Affiliations: | 2003 | University of Missouri - Kansas City, USA |
Area:
Cell Biology, General BiophysicsGoogle:
"Cole Haynes"Mean distance: (not calculated yet)
Parents
Sign in to add mentorAntony A. Cooper | grad student | 2003 | University of Missouri - Kansas City | |
(Degradation of misfolded proteins from the endoplasmic reticulum: Identification of a novel mechanism and prevention of cell death.) |
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Publications
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Dai CY, Ng CC, Hung GCC, et al. (2023) ATFS-1 counteracts mitochondrial DNA damage by promoting repair over transcription. Nature Cell Biology |
Uma Naresh N, Kim S, Shpilka T, et al. (2022) Mitochondrial genome recovery by ATFS-1 is essential for development after starvation. Cell Reports. 41: 111875 |
Haynes CM, Hekimi S. (2022) Mitochondrial dysfunction, aging, and the mitochondrial unfolded protein response in Caenorhabditis elegans. Genetics |
Statzer C, Meng J, Venz R, et al. (2022) ATF-4 and hydrogen sulfide signalling mediate longevity in response to inhibition of translation or mTORC1. Nature Communications. 13: 967 |
Yang Q, Liu P, Anderson NS, et al. (2022) LONP-1 and ATFS-1 sustain deleterious heteroplasmy by promoting mtDNA replication in dysfunctional mitochondria. Nature Cell Biology. 24: 181-193 |
Shpilka T, Du Y, Yang Q, et al. (2021) UPR scales mitochondrial network expansion with protein synthesis via mitochondrial import in Caenorhabditis elegans. Nature Communications. 12: 479 |
Ali L, Haynes CM. (2020) Mitochondrial translation, dynamics, and lysosomes combine to extend lifespan. The Journal of Cell Biology. 219 |
Anderson NS, Haynes CM. (2020) Folding the Mitochondrial UPR into the Integrated Stress Response. Trends in Cell Biology. 30: 428-439 |
Milstone ZJ, Saheera S, Bourke LM, et al. (2020) Histone deacetylases 1 and 2 silence cryptic transcription to promote mitochondrial function during cardiogenesis. Science Advances. 6: eaax5150 |
Tremblay BP, Haynes CM. (2020) Mitochondrial distress call moves to the cytosol to trigger a response to stress. Nature. 579: 348-349 |