Jesse E. Hanson

Affiliations: 
Stanford University School of Medicine, Palo Alto, CA, United States 
Area:
Synaptic Physiology, Fragile X, Parkinsons
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"Jesse Hanson"
Mean distance: 13.45 (cluster 6)
 
SNBCP

Parents

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Dieter Jaeger grad student 2003 Emory
 (Synaptic integration of excitatory inputs in globus pallidus neurons.)
Yoland Smith grad student 2003 Emory
 (Synaptic integration of excitatory inputs in globus pallidus neurons.)
Daniel V. Madison post-doc Stanford
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Publications

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Volgraf M, Sellers BD, Jiang Y, et al. (2016) Discovery of GluN2A-Selective NMDA Receptor Positive Allosteric Modulators (PAMs): Tuning Deactivation Kinetics via Structure-Based Design. Journal of Medicinal Chemistry
Hackos DH, Lupardus PJ, Grand T, et al. (2016) Positive Allosteric Modulators of GluN2A-Containing NMDARs with Distinct Modes of Action and Impacts on Circuit Function. Neuron
Hanson JE, Pare JF, Deng L, et al. (2015) Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease. Neurobiology of Disease. 74: 254-62
Orr AL, Hanson JE, Li D, et al. (2014) β-Amyloid inhibits E-S potentiation through suppression of cannabinoid receptor 1-dependent synaptic disinhibition. Neuron. 82: 1334-45
Hanson JE, Meilandt WJ, Gogineni A, et al. (2014) Chronic GluN2B antagonism disrupts behavior in wild-type mice without protecting against synapse loss or memory impairment in Alzheimer's disease mouse models. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 8277-88
Pozniak CD, Sengupta Ghosh A, Gogineni A, et al. (2013) Dual leucine zipper kinase is required for excitotoxicity-induced neuronal degeneration. The Journal of Experimental Medicine. 210: 2553-67
Hanson JE, La H, Plise E, et al. (2013) SAHA enhances synaptic function and plasticity in vitro but has limited brain availability in vivo and does not impact cognition. Plos One. 8: e69964
Hanson JE, Deng L, Hackos DH, et al. (2013) Histone deacetylase 2 cell autonomously suppresses excitatory and enhances inhibitory synaptic function in CA1 pyramidal neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 5924-9
Hanson JE, Weber M, Meilandt WJ, et al. (2013) GluN2B antagonism affects interneurons and leads to immediate and persistent changes in synaptic plasticity, oscillations, and behavior. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 38: 1221-33
Selcher JC, Xu W, Hanson JE, et al. (2012) Glutamate receptor subunit GluA1 is necessary for long-term potentiation and synapse unsilencing, but not long-term depression in mouse hippocampus. Brain Research. 1435: 8-14
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