Lawrence F. Brass
Affiliations: | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Pharmacology, Cell BiologyGoogle:
"Lawrence Brass"
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Publications
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| Kim OV, Litvinov RI, Gagne AL, et al. (2023) Megakaryocyte-induced contraction of plasma clots: Cellular mechanisms and structural mechanobiology. Blood |
| Gupta S, Cooper M, Zhao X, et al. (2023) A regulatory node involving Gα, PLCβ, and RGS proteins modulates platelet reactivity to critical agonists. Journal of Thrombosis and Haemostasis : Jth |
| DeCortin ME, Brass LF, Diamond SL. (2020) Core and shell platelets of a thrombus: A new microfluidic assay to study mechanics and biochemistry. Research and Practice in Thrombosis and Haemostasis. 4: 1158-1166 |
| Gupta S, Konradt C, Corken A, et al. (2020) Hemostasis vs. homeostasis: Platelets are essential for preserving vascular barrier function in the absence of injury or inflammation. Proceedings of the National Academy of Sciences of the United States of America |
| DeHelian DJ, Gupta S, Wu J, et al. (2020) RGS10 and RGS18 differentially limit platelet activation, promote platelet production, and prolong platelet survival. Blood |
| Chen X, Gupta S, Cooper M, et al. (2020) GRK6 regulates the hemostatic response to injury through its rate-limiting effects on GPCR signaling in platelets. Blood Advances. 4: 76-86 |
| Storrie B, Rhee SW, Pokrovskaya ID, et al. (2019) Platelet Activation State Intermixing in a Venous Puncture Model Indicates Novel Patterns of Thrombus Formation. Blood. 134: 9 |
| Tomaiuolo M, Matzko CN, Poventud-Fuentes I, et al. (2019) Interrelationships between structure and function during the hemostatic response to injury. Proceedings of the National Academy of Sciences of the United States of America |
| Ma P, Gupta S, Sampietro S, et al. (2018) RGS10 shapes the hemostatic response to injury through its differential effects on intracellular signaling by platelet agonists. Blood Advances. 2: 2145-2155 |
| Mirramezani M, Herbig BA, Stalker TJ, et al. (2018) Platelet packing density is an independent regulator of the hemostatic response to injury. Journal of Thrombosis and Haemostasis : Jth |