Indroneal Banerjee, Ph.D.
Affiliations: | 2009 | Biological Sciences | University of South Carolina, Columbia, SC |
Area:
Molecular Biology, Cell BiologyGoogle:
"Indroneal Banerjee"Parents
Sign in to add mentor| Troy A. Baudino | grad student | 2009 | University of South Carolina | |
| (Understanding the dynamic interactions of the cells in the heart.) | ||||
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Publications
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| Lange S, Banerjee I, Carrion K, et al. (2019) miR-486 is modulated by stretch and increases ventricular growth. Jci Insight |
| Moore-Morris T, Cattaneo P, Guimarães-Camboa N, et al. (2017) Infarct Fibroblasts Do Not Derive From Bone Marrow Lineages. Circulation Research |
| Klos M, Mundada L, Banerjee I, et al. (2017) Altered myocyte contractility and calcium homeostasis in alpha-myosin heavy chain point mutations linked to familial dilated cardiomyopathy. Archives of Biochemistry and Biophysics |
| Liang X, Zhang Q, Cattaneo P, et al. (2015) Transcription factor ISL1 is essential for pacemaker development and function. The Journal of Clinical Investigation |
| Lao DH, Esparza MC, Bremner SN, et al. (2015) Lmo7 is dispensable for skeletal muscle and cardiac function. American Journal of Physiology. Cell Physiology. ajpcell.00177.2015 |
| Wei C, Qiu J, Zhou Y, et al. (2015) Repression of the Central Splicing Regulator RBFox2 Is Functionally Linked to Pressure Overload-Induced Heart Failure. Cell Reports |
| Banerjee I, Carrion K, Serrano R, et al. (2015) Cyclic stretch of embryonic cardiomyocytes increases proliferation, growth, and expression while repressing Tgf-β signaling. Journal of Molecular and Cellular Cardiology. 79: 133-44 |
| Chapman MA, Zhang J, Banerjee I, et al. (2014) Disruption of both nesprin 1 and desmin results in nuclear anchorage defects and fibrosis in skeletal muscle. Human Molecular Genetics. 23: 5879-92 |
| Moore-Morris T, Guimarães-Camboa N, Banerjee I, et al. (2014) Resident fibroblast lineages mediate pressure overload-induced cardiac fibrosis. The Journal of Clinical Investigation. 124: 2921-34 |
| Banerjee I, Zhang J, Moore-Morris T, et al. (2014) Targeted ablation of nesprin 1 and nesprin 2 from murine myocardium results in cardiomyopathy, altered nuclear morphology and inhibition of the biomechanical gene response. Plos Genetics. 10: e1004114 |