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David M. Livingston, M.D.

Affiliations: 
Dana-Farber Cancer Institute, Boston, MA, United States 
Area:
Medical Genetics, Hematology, Oncology
Website:
https://www.dana-farber.org/find-a-doctor/david-m-livingston/
Google:
"David M. Livingston"
Bio:

http://www.nasonline.org/member-directory/members/1873.html

Mean distance: 21.97 (cluster 51)
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Parents

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Philip Leder post-doc

Children

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James A. DeCaprio grad student Dana-Farber Cancer Institute (Microtree)
Sarah James Hill grad student 2014 Harvard
Maria Guiseppina Baratta post-doc Dana-Farber Cancer Institute
Sharon Cantor post-doc University Of Massachusetts Med Sch Worcester
Junjie Chen post-doc Dana-Farber Cancer Institute and Harvard Medical School
Ronny Drapkin post-doc University of Pennsylvania Perelman School of Medicine
Marc Ewen post-doc Dana-Farber Cancer Institute
Jim Figge post-doc Dana-Farber Cancer Institute
Anthony G. Letai post-doc Dana-Farber Cancer Institute (Cell Biology Tree)
Ralph Scully post-doc Beth Israel Deaconess Medical Center and Harvard Medical School (Cell Biology Tree)
William Raj Sellers post-doc Dana-Farber Cancer Institute
Daniel Silver post-doc Thomas Jefferson
Brian Wolpin post-doc Dana-Farber Cancer Institute
Myles Brown post-doc 1982-1986 (Cell Biology Tree)
William G. Kaelin post-doc 1990 Dana-Farber Cancer Institute (Cell Biology Tree)
Andrew L. Kung post-doc 1999-2002 Memorial Sloan Kettering Cancer Center

Collaborators

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BETA: Related publications

Publications

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Hatchi E, Livingston DM. (2020) Opening a Door to PARP Inhibitor-Induced Lethality in HR-Proficient Human Tumor Cells. Cancer Cell. 37: 139-140
Vohhodina J, Toomire KJ, Petit SA, et al. (2020) RAP80 and BRCA1 PARsylation protect chromosome integrity by preventing retention of BRCA1-B/C complexes in DNA repair foci. Proceedings of the National Academy of Sciences of the United States of America
Wang H, Xiang D, Liu B, et al. (2019) Inadequate DNA Damage Repair Promotes Mammary Transdifferentiation, Leading to BRCA1 Breast Cancer. Cell. 178: 135-151.e19
Li AG, Murphy EC, Culhane AC, et al. (2018) BRCA1-IRIS promotes human tumor progression through PTEN blockade and HIF-1α activation. Proceedings of the National Academy of Sciences of the United States of America
Hill SJ, Mordes DA, Cameron LA, et al. (2016) Two familial ALS proteins function in prevention/repair of transcription-associated DNA damage. Proceedings of the National Academy of Sciences of the United States of America
Liu JF, Palakurthi S, Zeng Q, et al. (2016) Establishment of patient-derived tumor xenograft models of epithelial ovarian cancer for pre-clinical evaluation of novel therapeutics. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research
Wang H, Bierie B, Li AG, et al. (2016) BRCA1/FANCD2/BRG1-Driven DNA Repair Stabilizes the Differentiation State of Human Mammary Epithelial Cells. Molecular Cell
De Nicolo A, Haibe-Kains B, Taşan M, et al. (2016) Analysis of BRCA1-related functional associations in sporadic triple negative breast cancer: A network-based approach. Journal of Clinical Oncology. 34: 1070-1070
Brown M, Chinnaiyan A, Farsetti A, et al. (2015) 26th Pezcoller symposium: cancers driven by hormones. Cancer Research. 75: 1177-80
Hatchi E, Skourti-Stathaki K, Ventz S, et al. (2015) BRCA1 recruitment to transcriptional pause sites is required for R-loop-driven DNA damage repair. Molecular Cell. 57: 636-47
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