David M. Livingston, M.D.
Affiliations: | Dana-Farber Cancer Institute, Boston, MA, United States |
Area:
Medical Genetics, Hematology, OncologyWebsite:
https://www.dana-farber.org/find-a-doctor/david-m-livingston/Google:
"David M. Livingston"Bio:
http://www.nasonline.org/member-directory/members/1873.html
Mean distance: 21.97 (cluster 51)
Children
Sign in to add trainee| James A. DeCaprio | grad student | Dana-Farber Cancer Institute (Microtree) | |
| Sarah James Hill | grad student | 2014 | Harvard |
| Maria Guiseppina Baratta | post-doc | Dana-Farber Cancer Institute | |
| Sharon Cantor | post-doc | University Of Massachusetts Med Sch Worcester | |
| Junjie Chen | post-doc | Dana-Farber Cancer Institute and Harvard Medical School | |
| Ronny Drapkin | post-doc | University of Pennsylvania Perelman School of Medicine | |
| Marc Ewen | post-doc | Dana-Farber Cancer Institute | |
| Jim Figge | post-doc | Dana-Farber Cancer Institute | |
| Anthony G. Letai | post-doc | Dana-Farber Cancer Institute (Cell Biology Tree) | |
| Ralph Scully | post-doc | Beth Israel Deaconess Medical Center and Harvard Medical School (Cell Biology Tree) | |
| William Raj Sellers | post-doc | Dana-Farber Cancer Institute | |
| Daniel Silver | post-doc | Thomas Jefferson | |
| Brian Wolpin | post-doc | Dana-Farber Cancer Institute | |
| Myles Brown | post-doc | 1982-1986 | (Cell Biology Tree) |
| William G. Kaelin | post-doc | 1990 | Dana-Farber Cancer Institute (Cell Biology Tree) |
BETA: Related publications
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Publications
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| Hatchi E, Livingston DM. (2020) Opening a Door to PARP Inhibitor-Induced Lethality in HR-Proficient Human Tumor Cells. Cancer Cell. 37: 139-140 |
| Vohhodina J, Toomire KJ, Petit SA, et al. (2020) RAP80 and BRCA1 PARsylation protect chromosome integrity by preventing retention of BRCA1-B/C complexes in DNA repair foci. Proceedings of the National Academy of Sciences of the United States of America |
| Wang H, Xiang D, Liu B, et al. (2019) Inadequate DNA Damage Repair Promotes Mammary Transdifferentiation, Leading to BRCA1 Breast Cancer. Cell. 178: 135-151.e19 |
| Li AG, Murphy EC, Culhane AC, et al. (2018) BRCA1-IRIS promotes human tumor progression through PTEN blockade and HIF-1α activation. Proceedings of the National Academy of Sciences of the United States of America |
| Hill SJ, Mordes DA, Cameron LA, et al. (2016) Two familial ALS proteins function in prevention/repair of transcription-associated DNA damage. Proceedings of the National Academy of Sciences of the United States of America |
| Liu JF, Palakurthi S, Zeng Q, et al. (2016) Establishment of patient-derived tumor xenograft models of epithelial ovarian cancer for pre-clinical evaluation of novel therapeutics. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research |
| Wang H, Bierie B, Li AG, et al. (2016) BRCA1/FANCD2/BRG1-Driven DNA Repair Stabilizes the Differentiation State of Human Mammary Epithelial Cells. Molecular Cell |
| De Nicolo A, Haibe-Kains B, Taşan M, et al. (2016) Analysis of BRCA1-related functional associations in sporadic triple negative breast cancer: A network-based approach. Journal of Clinical Oncology. 34: 1070-1070 |
| Brown M, Chinnaiyan A, Farsetti A, et al. (2015) 26th Pezcoller symposium: cancers driven by hormones. Cancer Research. 75: 1177-80 |
| Hatchi E, Skourti-Stathaki K, Ventz S, et al. (2015) BRCA1 recruitment to transcriptional pause sites is required for R-loop-driven DNA damage repair. Molecular Cell. 57: 636-47 |