Hibiki Kawamata
Affiliations: | 1999-2001 | Neurology | Massachusetts General Hospital, Boston, MA |
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Parents
Sign in to add mentorPamela J. McLean | research assistant | 1999-2001 | MGH & Harvard Medical School |
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Publications
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McAvoy K, Kawamata H. (2019) Glial mitochondrial function and dysfunction in health and neurodegeneration. Molecular and Cellular Neurosciences. 101: 103417 |
Hayes LR, Asress SA, Li Y, et al. (2019) Distal denervation in the SOD1 knockout mouse correlates with loss of mitochondria at the motor nerve terminal. Experimental Neurology |
Anderson CJ, Bredvik K, Burstein SR, et al. (2019) ALS/FTD mutant CHCHD10 mice reveal a tissue-specific toxic gain-of-function and mitochondrial stress response. Acta Neuropathologica |
Palomo GM, Granatiero V, Kawamata H, et al. (2018) Parkin is a disease modifier in the mutant SOD1 mouse model of ALS. Embo Molecular Medicine |
Kawamata H, Manfredi G. (2018) Correction: Proteinopathies and OXPHOS dysfunction in neurodegenerative diseases. The Journal of Cell Biology. 217: 429 |
Kawamata H, Manfredi G. (2017) Proteinopathies and OXPHOS dysfunction in neurodegenerative diseases. The Journal of Cell Biology. 216: 3917-3929 |
Burstein SR, Valsecchi F, Kawamata H, et al. (2017) In vitro and in vivo studies of the ALS-FTLD protein CHCHD10 reveal novel mitochondrial topology and protein interactions. Human Molecular Genetics |
Kawamata H, Peixoto P, Konrad C, et al. (2017) Mutant TDP-43 does not impair mitochondrial bioenergetics in vitro and in vivo. Molecular Neurodegeneration. 12: 37 |
Doczi J, Torocsik B, Echaniz-Laguna A, et al. (2016) Alterations in voltage-sensing of the mitochondrial permeability transition pore in ANT1-deficient cells. Scientific Reports. 6: 26700 |
Manfredi G, Kawamata H. (2016) Mitochondria and endoplasmic reticulum crosstalk in amyotrophic lateral sclerosis. Neurobiology of Disease. 90: 35-42 |