Smrithi Prem
Affiliations: | Neuroscience | Rutgers-Robert Wood Johnson Medical School |
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| Prem S, Dev B, Peng C, et al. (2024) Dysregulation of mTOR signaling mediates common neurite and migration defects in both idiopathic and 16p11.2 deletion autism neural precursor cells. Elife. 13 |
| Connacher R, Williams M, Prem S, et al. (2022) Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports. 17: 1786 |
| Connacher R, Williams M, Prem S, et al. (2022) Autism NPCs from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports |
| Prem S, Millonig JH, DiCicco-Bloom E. (2020) Dysregulation of Neurite Outgrowth and Cell Migration in Autism and Other Neurodevelopmental Disorders. Advances in Neurobiology. 25: 109-153 |
| Turkalj L, Mehta M, Matteson P, et al. (2020) Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons. Advances in Neurobiology. 25: 79-107 |
| Williams M, Prem S, Zhou X, et al. (2018) Rapid Detection of Neurodevelopmental Phenotypes in Human Neural Precursor Cells (NPCs). Journal of Visualized Experiments : Jove |
| Genestine M, Lin L, Durens M, et al. (2015) Engrailed-2 (En2) deletion produces multiple neurodevelopmental defects in monoamine systems, forebrain structures and neurogenesis and behavior. Human Molecular Genetics |