Debora Masini, Ph.D

2019-2021 Neuroscience University of Copenhagen, København, Denmark 
 2022- Neuroscience Stockholm University, Stockholm, Stockholms län, Sweden 
"Debora Masini"
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Smedler E, Louhivuori L, Romanov RA, et al. (2022) Disrupted gene expression perturbs spontaneous Ca activity causing abnormal brain development and increased anxiety. Proceedings of the National Academy of Sciences of the United States of America. 119
Masini D, Kiehn O. (2022) Targeted activation of midbrain neurons restores locomotor function in mouse models of parkinsonism. Nature Communications. 13: 504
Masini D, Kiehn O. (2022) Targeted activation of midbrain neurons restores locomotor function in mouse models of parkinsonism Nature Communications
Masini D, Plewnia C, Bertho M, et al. (2021) A Guide to the Generation of a 6-Hydroxydopamine Mouse Model of Parkinson's Disease for the Study of Non-Motor Symptoms. Biomedicines. 9
Plewnia C, Masini D, Fisone G. (2020) P.327 Non-motor complications induced by dopamine replacement therapy in a mouse model of Parkinson's disease European Neuropsychopharmacology. 31: S67
Masini D, Bonito-Oliva A, Bertho M, et al. (2018) Inhibition of mTORC1 Signaling Reverts Cognitive and Affective Deficits in a Mouse Model of Parkinson's Disease. Frontiers in Neurology. 9: 208
Caggiano V, Leiras R, Goñi-Erro H, et al. (2018) Midbrain circuits that set locomotor speed and gait selection. Nature
di Val Cervo PR, Romanov RA, Spigolon G, et al. (2017) Induction of functional dopamine neurons from human astrocytes in vitro and mouse astrocytes in a Parkinson's disease model. Nature Biotechnology
Masini D, Lopes-Aguiar C, Bonito-Oliva A, et al. (2017) The histamine H3 receptor antagonist thioperamide rescues circadian rhythm and memory function in experimental parkinsonism. Translational Psychiatry. 7
Bonito-Oliva A, Masini D, Fisone G. (2014) A mouse model of non-motor symptoms in Parkinson's disease: focus on pharmacological interventions targeting affective dysfunctions. Frontiers in Behavioral Neuroscience. 8: 290
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