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Sidharth Tyagi

Affiliations: 
Yale University, New Haven, CT 
Area:
Sodium Channels, Calcium Channels
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"Sidharth Tyagi"
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Roger A. Bannister grad student
Stephen G. Waxman grad student

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Tyagi S, Higerd-Rusli GP, Akin EJ, et al. (2024) Real-time imaging of axonal membrane protein life cycles. Nature Protocols
Ghovanloo MR, Effraim PR, Tyagi S, et al. (2024) Functionally-selective inhibition of threshold sodium currents and excitability in dorsal root ganglion neurons by cannabinol. Communications Biology. 7: 120
Tyagi S, Higerd-Rusli GP, Ghovanloo MR, et al. (2024) Compartment-specific regulation of Na1.7 in sensory neurons after acute exposure to TNF-α. Cell Reports. 43: 113685
Ghovanloo MR, Tyagi S, Zhao P, et al. (2023) Sodium currents in naïve mouse dorsal root ganglion neurons: No major differences between sexes. Channels (Austin, Tex.). 18: 2289256
Tyagi S, Sarveswaran N, Higerd-Rusli GP, et al. (2023) Conserved but not critical: Trafficking and function of Na1.7 are independent of highly conserved polybasic motifs. Frontiers in Molecular Neuroscience. 16: 1161028
Higerd-Rusli GP, Tyagi S, Baker CA, et al. (2023) Inflammation differentially controls transport of depolarizing Nav versus hyperpolarizing Kv channels to drive rat nociceptor activity. Proceedings of the National Academy of Sciences of the United States of America. 120: e2215417120
Baker CA, Tyagi S, Higerd-Rusli GP, et al. (2023) Paclitaxel effects on axonal localization and vesicular trafficking of Na1.8. Frontiers in Molecular Neuroscience. 16: 1130123
Ghovanloo MR, Tyagi S, Zhao P, et al. (2023) High-throughput combined voltage-clamp/current-clamp analysis of freshly isolated neurons. Cell Reports Methods. 3: 100385
Higerd-Rusli GP, Tyagi S, Liu S, et al. (2022) The fates of internalized Na1.7 channels in sensory neurons: Retrograde cotransport with other ion channels, axon-specific recycling, and degradation. The Journal of Biological Chemistry. 299: 102816
Grosso BJ, Kramer AA, Tyagi S, et al. (2022) Complex effects on Ca2.1 channel gating caused by a CACNA1A variant associated with a severe neurodevelopmental disorder. Scientific Reports. 12: 9186
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