Stephen G. Waxman

Affiliations: 
Yale University, New Haven, CT 
Area:
pain, ion channels
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"Stephen Waxman"
Mean distance: 12.35 (cluster 6)
 
SNBCP
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Publications

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Lu W, Cheng X, Chen J, et al. (2021) A Buthus martensii Karsch scorpion sting targets Nav1.7 in mice and mimics a phenotype of human chronic pain. Pain
Gualdani R, Yuan JH, Effraim PR, et al. (2021) Trigeminal Neuralgia TRPM8 Mutation: Enhanced Activation, Basal [Ca] and Menthol Response. Neurology. Genetics. 7: e550
Xenakis MN, Kapetis D, Yang Y, et al. (2021) Hydropathicity-based prediction of pain-causing NaV1.7 variants. Bmc Bioinformatics. 22: 212
Benson CA, Olson KL, Patwa S, et al. (2021) Conditional RAC1 knockout in motor neurons restores H-reflex rate-dependent depression after spinal cord injury. Scientific Reports. 11: 7838
Xenakis MN, Kapetis D, Yang Y, et al. (2021) Non-extensitivity and criticality of atomic hydropathicity around a voltage-gated sodium channel's pore: a modeling study. Journal of Biological Physics
Akin EJ, Alsaloum M, Higerd GP, et al. (2021) Paclitaxel increases axonal localization and vesicular trafficking of Nav1.7. Brain : a Journal of Neurology
Honmou O, Yamashita T, Morita T, et al. (2021) Intravenous infusion of auto serum-expanded autologous mesenchymal stem cells in spinal cord injury patients: 13 case series. Clinical Neurology and Neurosurgery. 203: 106565
Almomani R, Marchi M, Sopacua M, et al. (2021) Correction: Evaluation of molecular inversion probe versus TruSeq® custom methods for targeted next-generation sequencing. Plos One. 16: e0248250
Chen L, Wimalasena NK, Shim J, et al. (2020) Two independent mouse lines carrying the Nav1.7-I228M gain-of-function variant display DRG neuron hyperexcitability but a minimal pain phenotype. Pain
Cheng X, Choi JS, Waxman SG, et al. (2020) Mini-review - Sodium channels and beyond in peripheral nerve disease: Modulation by cytokines and their effector protein kinases. Neuroscience Letters. 135446
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