Masanobu Kano

Affiliations: 
Neurophysiology The University of Tokyo, International Research Center for Neurointelligence 
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"Masanobu Kano"
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Hoang H, Tsutsumi S, Matsuzaki M, et al. (2023) Dynamic organization of cerebellar climbing fiber response and synchrony in multiple functional components reduces dimensions for reinforcement learning. Elife. 12
Ikezoe K, Hidaka N, Manita S, et al. (2023) Cerebellar climbing fibers multiplex movement and reward signals during a voluntary movement task in mice. Communications Biology. 6: 924
Okuno Y, Sakoori K, Matsuyama K, et al. (2023) PTPδ is a presynaptic organizer for the formation and maintenance of climbing fiber to Purkinje cell synapses in the developing cerebellum. Frontiers in Molecular Neuroscience. 16: 1206245
Obi-Nagata K, Suzuki N, Miyake R, et al. (2023) Distorted neurocomputation by a small number of extra-large spines in psychiatric disorders. Science Advances. 9: eade5973
Singh M, Sapkota K, Sakimura K, et al. (2023) Maturation of GABAergic Synaptic Transmission from Neocortical Parvalbumin Interneurons Involves NMDA receptor Recruitment of Cav2.1 Channels. Neuroscience
Itami C, Uesaka N, Huang JY, et al. (2022) Endocannabinoid-dependent formation of columnar axonal projection in the mouse cerebral cortex. Proceedings of the National Academy of Sciences of the United States of America. 119: e2122700119
Harbers M, Nakao H, Watanabe T, et al. (2022) mGluR5 Is Substitutable for mGluR1 in Cerebellar Purkinje Cells for Motor Coordination, Developmental Synapse Elimination, and Motor Learning. Cells. 11
Sakamoto M, Inoue M, Takeuchi A, et al. (2022) A Flp-dependent G-CaMP9a transgenic mouse for neuronal imaging . Cell Reports Methods. 2: 100168
Lai ESK, Nakayama H, Miyazaki T, et al. (2021) An Autism-Associated Neuroligin-3 Mutation Affects Developmental Synapse Elimination in the Cerebellum. Frontiers in Neural Circuits. 15: 676891
Toriumi K, Berto S, Koike S, et al. (2021) Combined glyoxalase 1 dysfunction and vitamin B6 deficiency in a schizophrenia model system causes mitochondrial dysfunction in the prefrontal cortex. Redox Biology. 45: 102057
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