Hisako Nakayama
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| Harachi M, Masui K, Shimizu E, et al. (2024) DNA hypomethylator phenotype reprograms glutamatergic network in receptor tyrosine kinase gene-mutated glioblastoma. Acta Neuropathologica Communications. 12: 40 |
| Lai ESK, Nakayama H, Miyazaki T, et al. (2021) An Autism-Associated Neuroligin-3 Mutation Affects Developmental Synapse Elimination in the Cerebellum. Frontiers in Neural Circuits. 15: 676891 |
| Nagumo Y, Ueta Y, Nakayama H, et al. (2020) Tonic GABAergic Inhibition Is Essential for Nerve Injury-Induced Afferent Remodeling in the Somatosensory Thalamus and Ectopic Sensations. Cell Reports. 31: 107797 |
| Sakai Y, Kassai H, Nakayama H, et al. (2019) Hyperactivation of mTORC1 disrupts cellular homeostasis in cerebellar Purkinje cells. Scientific Reports. 9: 2799 |
| Nakayama H, Abe M, Morimoto C, et al. (2018) Microglia permit climbing fiber elimination by promoting GABAergic inhibition in the developing cerebellum. Nature Communications. 9: 2830 |
| Matsumoto-Makidono Y, Nakayama H, Yamasaki M, et al. (2016) Ionic Basis for Membrane Potential Resonance in Neurons of the Inferior Olive. Cell Reports |
| Piochon C, Kloth AD, Grasselli G, et al. (2015) Corrigendum: Cerebellar plasticity and motor learning deficits in a copy-number variation mouse model of autism. Nature Communications. 6: 6014 |
| Piochon C, Kloth AD, Grasselli G, et al. (2014) Cerebellar plasticity and motor learning deficits in a copy-number variation mouse model of autism. Nature Communications. 5: 5586 |
| Kawamura Y, Nakayama H, Hashimoto K, et al. (2013) Spike timing-dependent selective strengthening of single climbing fibre inputs to Purkinje cells during cerebellar development. Nature Communications. 4: 2732 |
| Kano M, Nakayama H, Hashimoto K, et al. (2013) Calcium-dependent regulation of climbing fibre synapse elimination during postnatal cerebellar development. The Journal of Physiology. 591: 3151-8 |