Kristina Lorenz

1999-2015 Pharmacology and Toxicology University of Würzburg, Würzburg, Bayern, Germany 
"Kristina Lorenz"
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Martin J. Lohse grad student 1999-2015 University of Wurzburg
 (graduate student, postdoc, assoc prof. with intervening periods at Rochester, NY, and Dresden, Germany)
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Rodríguez-Soacha DA, Steinmüller SAM, Işbilir A, et al. (2022) Development of an Indole-Amide-Based Photoswitchable Cannabinoid Receptor Subtype 1 (CBR) "Cis-On" Agonist. Acs Chemical Neuroscience. 13: 2410-2435
Lee J, Olivieri C, Ong C, et al. (2022) Raf Kinase Inhibitory Protein regulates the cAMP-dependent protein kinase signaling pathway through a positive feedback loop. Proceedings of the National Academy of Sciences of the United States of America. 119: e2121867119
Lorenz K, Rosner MR. (2022) Harnessing RKIP to Combat Heart Disease and Cancer. Cancers. 14
Tomasovic A, Brand T, Schanbacher C, et al. (2020) Interference with ERK-dimerization at the nucleocytosolic interface targets pathological ERK1/2 signaling without cardiotoxic side-effects. Nature Communications. 11: 1733
Maimari T, Krasel C, Bünemann M, et al. (2019) The N-termini of GRK2 and GRK3 simulate the stimulating effects of RKIP on β-adrenoceptors. Biochemical and Biophysical Research Communications
Jochmann S, Elkenani M, Mohamed BA, et al. (2019) Assessing the role of extracellular signal-regulated kinases 1 and 2 in volume overload-induced cardiac remodelling. Esc Heart Failure
Skinner JJ, Wang S, Lee J, et al. (2017) Conserved salt-bridge competition triggered by phosphorylation regulates the protein interactome. Proceedings of the National Academy of Sciences of the United States of America
Lorenz K, Rosner MR, Brand T, et al. (2017) RKIP - Lessons of a better way for β-adrenergic receptor activation in the heart. The Journal of Physiology
Brietz A, Schuch KV, Wangorsch G, et al. (2016) Analyzing ERK 1/2 signalling and targets. Molecular Biosystems
Nuber S, Zabel U, Lorenz K, et al. (2016) β-Arrestin biosensors reveal a rapid, receptor-dependent activation/deactivation cycle. Nature
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