Sarah Ann Busch
Affiliations: | 2009 | Case Western Reserve University, Cleveland Heights, OH, United States |
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Parents
Sign in to add mentorJerry Silver | grad student | 2009 | Case Western | |
(Cellular and molecular strategies to overcome macrophage-mediated axonal dieback after spinal cord injury.) |
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Publications
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Jones JM, DePaul M, Gregory CR, et al. (2018) Multipotent adult progenitor cells, but not TIMP3, increase tissue sparing and reduce urological complications following spinal cord injury. Journal of Neurotrauma |
Jones J, DePaul M, Zhu M, et al. (2017) MP41-06 MULTIPOTENT ADULT PROGENITOR CELL TREATMENT PROMOTES BLADDER FUNCTION RECOVERY AND DECREASES MORBIDITY AND DURATION OF URINARY TRACT INFECTION IN SPINAL CORD INJURED RATS Journal of Urology. 197 |
DePaul MA, Palmer M, Lang BT, et al. (2015) Intravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury. Scientific Reports. 5: 16795 |
Lang BT, Cregg JM, DePaul MA, et al. (2015) Modulation of the proteoglycan receptor PTPσ promotes recovery after spinal cord injury. Nature. 518: 404-8 |
Filous AR, Tran A, Howell CJ, et al. (2014) Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 16369-84 |
Reading JL, Yang JH, Sabbah S, et al. (2013) Clinical-grade multipotent adult progenitor cells durably control pathogenic T cell responses in human models of transplantation and autoimmunity. Journal of Immunology (Baltimore, Md. : 1950). 190: 4542-52 |
Busch SA, Hamilton JA, Horn KP, et al. (2011) Multipotent adult progenitor cells prevent macrophage-mediated axonal dieback and promote regrowth after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 944-53 |
Busch SA, Horn KP, Cuascut FX, et al. (2010) Adult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 255-65 |
Silver J, Horn KP, Busch SA, et al. (2010) Axonal regeneration: Role of the extracellular matrix and the glial scar Encyclopedia of Neuroscience. 1173-1180 |
Shen Y, Tenney AP, Busch SA, et al. (2009) PTPsigma is a receptor for chondroitin sulfate proteoglycan, an inhibitor of neural regeneration. Science (New York, N.Y.). 326: 592-6 |