Year |
Citation |
Score |
2024 |
Kim S, Phan S, Tran HT, Shaw TR, Shahmoradian SH, Ellisman MH, Veatch SL, Barmada SJ, Pappas SS, Dauer WT. TorsinA is essential for neuronal nuclear pore complex localization and maturation. Nature Cell Biology. PMID 39117796 DOI: 10.1038/s41556-024-01480-1 |
0.344 |
|
2023 |
Morrison LM, Huang H, Handler HP, Fu M, Bushart DD, Pappas SS, Orr HT, Shakkottai VG. Increased intrinsic membrane excitability is associated with hypertrophic olivary degeneration in spinocerebellar ataxia type 1. Biorxiv : the Preprint Server For Biology. PMID 37961407 DOI: 10.1101/2023.10.23.563657 |
0.424 |
|
2022 |
Yellajoshyula D, Opeyemi S, Dauer WT, Pappas SS. Genetic evidence of aberrant striatal synaptic maturation and secretory pathway alteration in a dystonia mouse model. Dystonia. 1. PMID 36874764 DOI: 10.3389/dyst.2022.10892 |
0.44 |
|
2020 |
Li J, Liang CC, Pappas SS, Dauer WT. TorsinB overexpression prevents abnormal twisting in DYT1 dystonia mouse models. Elife. 9. PMID 32202496 DOI: 10.7554/Elife.54285 |
0.377 |
|
2018 |
Pappas SS, Li J, LeWitt TM, Kim JK, Monani UR, Dauer WT. A cell autonomous torsinA requirement for cholinergic neuron survival and motor control. Elife. 7. PMID 30117805 DOI: 10.7554/Elife.36691 |
0.58 |
|
2018 |
Weisheit CE, Pappas SS, Dauer WT. Inherited dystonias: clinical features and molecular pathways. Handbook of Clinical Neurology. 147: 241-254. PMID 29325615 DOI: 10.1016/B978-0-444-63233-3.00016-6 |
0.334 |
|
2018 |
Pappas SS, Li J, LeWitt TM, Kim J, Monani UR, Dauer WT. Author response: A cell autonomous torsinA requirement for cholinergic neuron survival and motor control Elife. DOI: 10.7554/Elife.36691.024 |
0.425 |
|
2017 |
Pappas SS, Liang CC, Kim S, Rivera CO, Dauer WT. TorsinA dysfunction causes persistent neuronal nuclear pore defects. Human Molecular Genetics. PMID 29186574 DOI: 10.1093/Hmg/Ddx405 |
0.505 |
|
2017 |
DeSimone JC, Pappas SS, Febo M, Burciu RG, Shukla P, Colon-Perez LM, Dauer WT, Vaillancourt DE. Forebrain knock-out of torsinA reduces striatal free-water and impairs whole-brain functional connectivity in a symptomatic mouse model of DYT1 dystonia. Neurobiology of Disease. PMID 28673740 DOI: 10.1016/J.Nbd.2017.06.015 |
0.365 |
|
2015 |
Pappas SS, Darr K, Holley SM, Cepeda C, Mabrouk OS, Wong JM, LeWitt TM, Paudel R, Houlden H, Kennedy RT, Levine MS, Dauer WT. Forebrain deletion of the dystonia protein torsinA causes dystonic-like movements and loss of striatal cholinergic neurons. Elife. 4. PMID 26052670 DOI: 10.7554/Elife.08352 |
0.444 |
|
2015 |
Pappas SS, Darr K, Holley SM, Cepeda C, Mabrouk OS, Wong JT, LeWitt TM, Paudel R, Houlden H, Kennedy RT, Levine MS, Dauer WT. Author response: Forebrain deletion of the dystonia protein torsinA causes dystonic-like movements and loss of striatal cholinergic neurons Elife. DOI: 10.7554/Elife.08352.023 |
0.491 |
|
2014 |
Luna-Cancalon K, Sikora KM, Pappas SS, Singh V, Wulff H, Paulson HL, Burmeister M, Shakkottai VG. Alterations in cerebellar physiology are associated with a stiff-legged gait in Atcay(ji-hes) mice. Neurobiology of Disease. 67: 140-8. PMID 24727095 DOI: 10.1016/J.Nbd.2014.03.020 |
0.532 |
|
2012 |
Simkins TJ, Janis KL, McClure AK, Behrouz B, Pappas SS, Lehner A, Kaminski NE, Goudreau JL, Lookingland KJ, Kaplan BL. Comparison of the D2 receptor regulation and neurotoxicant susceptibility of nigrostriatal dopamine neurons in wild-type and CB1/CB2 receptor knockout mice. Journal of Neuroimmune Pharmacology : the Official Journal of the Society On Neuroimmune Pharmacology. 7: 533-8. PMID 22639229 DOI: 10.1007/S11481-012-9375-Y |
0.631 |
|
2012 |
Benskey M, Behrouz B, Sunryd J, Pappas SS, Baek SH, Huebner M, Lookingland KJ, Goudreau JL. Recovery of hypothalamic tuberoinfundibular dopamine neurons from acute toxicant exposure is dependent upon protein synthesis and associated with an increase in parkin and ubiquitin carboxy-terminal hydrolase-L1 expression. Neurotoxicology. 33: 321-31. PMID 22342763 DOI: 10.1016/J.Neuro.2012.02.001 |
0.76 |
|
2012 |
Benskey M, Behrouz B, Sunryd J, Pappas SS, Baek S, Huebner M, Lookingland KJ, Goudreau JL. Erratum to “Recovery of hypothalamic tuberoinfundibular dopamine neurons from acute toxicant exposure is dependent upon protein synthesis and associated with an increase in parkin and ubiquitin carboxy-terminal hydrolase-L1 expression” [Neurotoxicology 33 (2012) 321–331] Neurotoxicology. 33: 406. DOI: 10.1016/J.Neuro.2012.04.013 |
0.731 |
|
2011 |
Pappas SS, Kennedy T, Goudreau JL, Lookingland KJ. Opioid-mediated regulation of A11 diencephalospinal dopamine neurons: pharmacological evidence of activation by morphine. Neuropharmacology. 61: 614-21. PMID 21605572 DOI: 10.1016/J.Neuropharm.2011.05.002 |
0.756 |
|
2010 |
Pappas SS, Tiernan CT, Behrouz B, Jordan CL, Breedlove SM, Goudreau JL, Lookingland KJ. Neonatal androgen-dependent sex differences in lumbar spinal cord dopamine concentrations and the number of A11 diencephalospinal dopamine neurons. The Journal of Comparative Neurology. 518: 2423-36. PMID 20503420 DOI: 10.1002/Cne.22340 |
0.718 |
|
2008 |
Pappas SS, Behrouz B, Janis KL, Goudreau JL, Lookingland KJ. Lack of D2 receptor mediated regulation of dopamine synthesis in A11 diencephalospinal neurons in male and female mice. Brain Research. 1214: 1-10. PMID 18462709 DOI: 10.1016/J.Brainres.2008.03.010 |
0.712 |
|
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