Breset Walker, Ph.D. - Publications

Affiliations: 
2012 Weill Cornell Medical College, New York, NY, United States 

6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2012 Walker BA, Ji SJ, Jaffrey SR. Intra-axonal translation of RhoA promotes axon growth inhibition by CSPG. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 14442-7. PMID 23055514 DOI: 10.1523/JNEUROSCI.0176-12.2012  0.96
2012 Walker BA, Hengst U, Kim HJ, Jeon NL, Schmidt EF, Heintz N, Milner TA, Jaffrey SR. Reprogramming axonal behavior by axon-specific viral transduction. Gene Therapy. 19: 947-55. PMID 22278412 DOI: 10.1038/gt.2011.217  0.96
2011 El-Hattab AW, Bournat J, Eng PA, Wu JB, Walker BA, Stankiewicz P, Cheung SW, Brown CW. Microduplication of Xp11.23p11.3 with effects on cognition, behavior, and craniofacial development. Clinical Genetics. 79: 531-8. PMID 20662849 DOI: 10.1111/j.1399-0004.2010.01496.x  0.96
2010 Claus CP, Tsuru-Aoyagi K, Adwanikar H, Walker B, Manvelyan H, Whetstone W, Noble-Haeusslein LJ. Age is a determinant of leukocyte infiltration and loss of cortical volume after traumatic brain injury. Developmental Neuroscience. 32: 454-65. PMID 20847543 DOI: 10.1159/000316805  0.96
2009 Rivieccio MA, Brochier C, Willis DE, Walker BA, D'Annibale MA, McLaughlin K, Siddiq A, Kozikowski AP, Jaffrey SR, Twiss JL, Ratan RR, Langley B. HDAC6 is a target for protection and regeneration following injury in the nervous system. Proceedings of the National Academy of Sciences of the United States of America. 106: 19599-604. PMID 19884510 DOI: 10.1073/pnas.0907935106  0.96
2006 Potts MB, Koh SE, Whetstone WD, Walker BA, Yoneyama T, Claus CP, Manvelyan HM, Noble-Haeusslein LJ. Traumatic injury to the immature brain: inflammation, oxidative injury, and iron-mediated damage as potential therapeutic targets. Neurorx : the Journal of the American Society For Experimental Neurotherapeutics. 3: 143-53. PMID 16554253 DOI: 10.1016/j.nurx.2006.01.006  0.96
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