Vera Valakh - Publications

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14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2024 Wise DL, Escobedo-Lozoya Y, Valakh V, Gao EY, Bhonsle A, Lei QL, Cheng X, Greene SB, Van Hooser SD, Nelson SB. Prolonged activity-deprivation causes pre- and postsynaptic compensatory plasticity at neocortical excitatory synapses. Eneuro. PMID 38777611 DOI: 10.1523/ENEURO.0366-23.2024  0.435
2023 Valakh V, Wise D, Zhu XA, Sha M, Fok J, Van Hooser SD, Schectman R, Cepeda I, Kirk R, O'Toole SM, Nelson SB. A transcriptional constraint mechanism limits the homeostatic response to activity deprivation in mammalian neocortex. Elife. 12. PMID 36749029 DOI: 10.7554/eLife.74899  0.36
2020 Valakh V, Flyer-Adams JG. Intracellular Recordings of Postsynaptic Voltage Responses at the Drosophila Neuromuscular Junction. Methods in Molecular Biology (Clifton, N.J.). 2143: 159-168. PMID 32524479 DOI: 10.1007/978-1-0716-0585-1_12  0.333
2015 Valakh V, Frey E, Babetto E, Walker LJ, DiAntonio A. Cytoskeletal disruption activates the DLK/JNK pathway, which promotes axonal regeneration and mimics a preconditioning injury. Neurobiology of Disease. 77: 13-25. PMID 25726747 DOI: 10.1016/J.Nbd.2015.02.014  0.641
2015 Frey E, Valakh V, Karney-Grobe S, Shi Y, Milbrandt J, DiAntonio A. An in vitro assay to study induction of the regenerative state in sensory neurons. Experimental Neurology. 263: 350-63. PMID 25447942 DOI: 10.1016/J.Expneurol.2014.10.012  0.599
2014 Brace EJ, Wu C, Valakh V, DiAntonio A. SkpA restrains synaptic terminal growth during development and promotes axonal degeneration following injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 8398-410. PMID 24948796 DOI: 10.1523/Jneurosci.4715-13.2014  0.707
2014 Zheng X, Valakh V, Diantonio A, Ben-Shahar Y. Natural antisense transcripts regulate the neuronal stress response and excitability. Elife. 3: e01849. PMID 24642409 DOI: 10.7554/Elife.01849  0.568
2014 Zheng X, Valakh V, DiAntonio A, Ben-Shahar Y. Author response: Natural antisense transcripts regulate the neuronal stress response and excitability Elife. DOI: 10.7554/Elife.01849.021  0.543
2013 Valakh V, Walker LJ, Skeath JB, DiAntonio A. Loss of the spectraplakin short stop activates the DLK injury response pathway in Drosophila. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 17863-73. PMID 24198375 DOI: 10.1523/Jneurosci.2196-13.2013  0.67
2012 Graf ER, Valakh V, Wright CM, Wu C, Liu Z, Zhang YQ, DiAntonio A. RIM promotes calcium channel accumulation at active zones of the Drosophila neuromuscular junction. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 16586-96. PMID 23175814 DOI: 10.1523/Jneurosci.0965-12.2012  0.747
2012 Valakh V, Naylor SA, Berns DS, DiAntonio A. A large-scale RNAi screen identifies functional classes of genes shaping synaptic development and maintenance. Developmental Biology. 366: 163-71. PMID 22542760 DOI: 10.1016/J.Ydbio.2012.04.008  0.721
2011 Tian X, Li J, Valakh V, DiAntonio A, Wu C. Drosophila Rae1 controls the abundance of the ubiquitin ligase Highwire in post-mitotic neurons. Nature Neuroscience. 14: 1267-75. PMID 21874015 DOI: 10.1038/Nn.2922  0.76
2009 Viquez NM, Füger P, Valakh V, Daniels RW, Rasse TM, DiAntonio A. PP2A and GSK-3beta act antagonistically to regulate active zone development. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 11484-94. PMID 19759297 DOI: 10.1523/Jneurosci.5584-08.2009  0.58
2008 de Marchena J, Roberts AC, Middlebrooks PG, Valakh V, Yashiro K, Wilfley LR, Philpot BD. NMDA receptor antagonists reveal age-dependent differences in the properties of visual cortical plasticity. Journal of Neurophysiology. 100: 1936-48. PMID 18667547 DOI: 10.1152/Jn.90290.2008  0.338
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