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Ann M. Gregus, Ph.D. - Publications

Affiliations: 
2008 Weill Cornell Medical College, New York, NY, United States 
Area:
Pharmacology, Neuroscience Biology
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7 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Chen I, Murdaugh LB, Miliano C, Dong Y, Gregus AM, Buczynski MW. NAPE-PLD regulates specific baseline affective behaviors but is dispensable for inflammatory hyperalgesia. Neurobiology of Pain (Cambridge, Mass.). 14: 100135. PMID 38099275 DOI: 10.1016/j.ynpai.2023.100135  0.641
2018 Gregus AM, Buczynski MW, Dumlao DS, Norris PC, Rai G, Simeonov A, Maloney DJ, Jadhav A, Xu Q, Wei SC, Fitzsimmons BL, Dennis EA, Yaksh TL. Inhibition of Spinal 15-LOX-1 Attenuates TLR4-Dependent, NSAID-Unresponsive Hyperalgesia in Male Rats. Pain. PMID 30130298 DOI: 10.1097/J.Pain.0000000000001373  0.546
2013 Gregus AM, Dumlao DS, Wei SC, Norris PC, Catella LC, Meyerstein FG, Buczynski MW, Steinauer JJ, Fitzsimmons BL, Yaksh TL, Dennis EA. Systematic analysis of rat 12/15-lipoxygenase enzymes reveals critical role for spinal eLOX3 hepoxilin synthase activity in inflammatory hyperalgesia. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 27: 1939-49. PMID 23382512 DOI: 10.1096/Fj.12-217414  0.351
2012 Gregus AM, Doolen S, Dumlao DS, Buczynski MW, Takasusuki T, Fitzsimmons BL, Hua XY, Taylor BK, Dennis EA, Yaksh TL. Spinal 12-lipoxygenase-derived hepoxilin A3 contributes to inflammatory hyperalgesia via activation of TRPV1 and TRPA1 receptors. Proceedings of the National Academy of Sciences of the United States of America. 109: 6721-6. PMID 22493235 DOI: 10.1073/Pnas.1110460109  0.403
2010 Gregus AM, Inra CN, Giordano TP, Costa AC, Rajadhyaksha AM, Inturrisi CE. Spinal mediators that may contribute selectively to antinociceptive tolerance but not other effects of morphine as revealed by deletion of GluR5. Neuroscience. 169: 475-87. PMID 20359526 DOI: 10.1016/J.Neuroscience.2010.03.051  0.731
2010 Gregus AM, Tropea TF, Wang Y, Hauck SC, Costa AC, Rajadhyaksha AM, Inturrisi CE. Deletion of the GluR5 subunit of kainate receptors affects cocaine sensitivity and preference. Neuroscience Letters. 468: 186-9. PMID 19878705 DOI: 10.1016/J.Neulet.2009.10.071  0.647
2009 Bogulavsky JJ, Gregus AM, Kim PT, Costa AC, Rajadhyaksha AM, Inturrisi CE. Deletion of the glutamate receptor 5 subunit of kainate receptors affects the development of morphine tolerance. The Journal of Pharmacology and Experimental Therapeutics. 328: 579-87. PMID 18957577 DOI: 10.1124/Jpet.108.144121  0.387
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