| Year |
Citation |
Score |
| 2020 |
Wu Z, Li L, Xie F, Xu G, Dang D, Yang Q. Enhancing KCNQ channel activity improves neurobehavioral recovery after spinal cord injury. The Journal of Pharmacology and Experimental Therapeutics. PMID 31969383 DOI: 10.1124/Jpet.119.264010 |
0.425 |
|
| 2018 |
Li L, Li J, Zuo Y, Dang D, Frost JA, Yang Q. Activation of KCNQ channels prevents paclitaxel-induced peripheral neuropathy and associated neuropathic pain. The Journal of Pain : Official Journal of the American Pain Society. PMID 30471428 DOI: 10.1016/J.Jpain.2018.11.001 |
0.387 |
|
| 2018 |
Zhang H, Li Y, Yang Q, Liu XG, Dougherty PM. Morphological and Physiological Plasticity of Spinal Lamina II GABA Neurons Is Induced by Sciatic Nerve Chronic Constriction Injury in Mice. Frontiers in Cellular Neuroscience. 12: 143. PMID 29881336 DOI: 10.3389/Fncel.2018.00143 |
0.333 |
|
| 2017 |
Wu Z, Li L, Xie F, Du J, Zuo Y, Frost JA, Carlton SM, Walters ET, Yang Q. Activation of KCNQ channels suppresses spontaneous activity in DRG neurons and reduces chronic pain after spinal cord injury. Journal of Neurotrauma. PMID 28073317 DOI: 10.1089/Neu.2016.4789 |
0.621 |
|
| 2016 |
Bavencoffe A, Li Y, Wu Z, Yang Q, Herrera J, Kennedy EJ, Walters ET, Dessauer CW. Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 1660-8. PMID 26843647 DOI: 10.1523/Jneurosci.0895-15.2016 |
0.61 |
|
| 2014 |
Yang Q, Wu Z, Hadden JK, Odem MA, Zuo Y, Crook RJ, Frost JA, Walters ET. Persistent pain after spinal cord injury is maintained by primary afferent activity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 10765-9. PMID 25100607 DOI: 10.1523/Jneurosci.5316-13.2014 |
0.619 |
|
| 2013 |
Yin S, Luo J, Qian A, Du J, Yang Q, Zhou S, Yu W, Du G, Clark RB, Walters ET, Carlton SM, Hu H. Retinoids activate the irritant receptor TRPV1 and produce sensory hypersensitivity. The Journal of Clinical Investigation. 123: 3941-51. PMID 23925292 DOI: 10.1172/Jci66413 |
0.577 |
|
| 2013 |
Wu Z, Yang Q, Crook RJ, O'Neil RG, Walters ET. TRPV1 channels make major contributions to behavioral hypersensitivity and spontaneous activity in nociceptors after spinal cord injury. Pain. 154: 2130-41. PMID 23811042 DOI: 10.1016/J.Pain.2013.06.040 |
0.625 |
|
| 2010 |
Bedi SS, Yang Q, Crook RJ, Du J, Wu Z, Fishman HM, Grill RJ, Carlton SM, Walters ET. Chronic spontaneous activity generated in the somata of primary nociceptors is associated with pain-related behavior after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 14870-82. PMID 21048146 DOI: 10.1523/Jneurosci.2428-10.2010 |
0.617 |
|
| 2008 |
Li DP, Yang Q, Pan HM, Pan HL. Pre- and postsynaptic plasticity underlying augmented glutamatergic inputs to hypothalamic presympathetic neurons in spontaneously hypertensive rats. The Journal of Physiology. 586: 1637-47. PMID 18238817 DOI: 10.1113/Jphysiol.2007.149732 |
0.324 |
|
| 2007 |
Yang Q, Chen SR, Li DP, Pan HL. Kv1.1/1.2 channels are downstream effectors of nitric oxide on synaptic GABA release to preautonomic neurons in the paraventricular nucleus. Neuroscience. 149: 315-27. PMID 17869444 DOI: 10.1016/J.Neuroscience.2007.08.007 |
0.316 |
|
| 2007 |
Li DP, Yang Q. Membrane and synaptic properties of nucleus tractus solitarius neurons projecting to the caudal ventrolateral medulla. Autonomic Neuroscience : Basic & Clinical. 136: 69-81. PMID 17537680 DOI: 10.1016/J.Autneu.2007.04.006 |
0.308 |
|
| 2004 |
Wu ZZ, Guan BC, Li ZW, Yang Q, Liu CJ, Chen JG. Sustained potentiation by substance P of NMDA-activated current in rat primary sensory neurons Brain Research. 1010: 117-126. PMID 15126124 DOI: 10.1016/J.Brainres.2004.03.010 |
0.331 |
|
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