Year |
Citation |
Score |
2016 |
Ye W, Bushman J, Liman E. Unique Biophysical Properties of an Inward Proton Current that Mediates Sour Taste Transduction Biophysical Journal. 110: 481a. DOI: 10.1016/J.Bpj.2015.11.2573 |
0.494 |
|
2016 |
Ye W, Chang RB, Bushman JD, Tu Y, Mulhall E, Wilson CE, Cooper AJ, Chick WS, Hill-Eubanks D, Nelson MT, Kinnamon SC, Liman ER. Inhibition of KIR2.1 by Intracellular Acidification Contributes to Sour Taste Transduction Biophysical Journal. 110: 424a. DOI: 10.1016/J.Bpj.2015.11.2294 |
0.37 |
|
2015 |
Ye W, Chang RB, Bushman JD, Tu YH, Mulhall EM, Wilson CE, Cooper AJ, Chick WS, Hill-Eubanks DC, Nelson MT, Kinnamon SC, Liman ER. K+ channel KIR2.1 functions in tandem with proton influx to mediate sour taste transduction. Proceedings of the National Academy of Sciences of the United States of America. PMID 26627720 DOI: 10.1073/Pnas.1514282112 |
0.411 |
|
2013 |
Bushman JD, Zhou Q, Shyng SL. A Kir6.2 pore mutation causes inactivation of ATP-sensitive potassium channels by disrupting PIP2-dependent gating. Plos One. 8: e63733. PMID 23700433 DOI: 10.1371/journal.pone.0063733 |
0.75 |
|
2012 |
Shyng SL, Bushman JD, Pratt EB, Zhou Q. Molecular defects of ATP-sensitive potassium channels in congenital hyperinsulinism Monogenic Hyperinsulinemic Hypoglycemia Disorders. 21: 30-42. DOI: 10.1159/000334485 |
0.604 |
|
2010 |
Bushman JD, Gay JW, Tewson P, Stanley CA, Shyng SL. Characterization and functional restoration of a potassium channel Kir6.2 pore mutation identified in congenital hyperinsulinism. The Journal of Biological Chemistry. 285: 6012-23. PMID 20032456 DOI: 10.1074/jbc.M109.085860 |
0.783 |
|
2009 |
Bushman JD, Tewson PH, Shyng S. Rescue and Gating of a Disease Mutation at an M2 glycine in Kir6.2 of ATP-Sensitive Potassium (KATP) Channels Biophysical Journal. 96: 465a-466a. DOI: 10.1016/J.BPJ.2008.12.2397 |
0.764 |
|
2008 |
Abdulhadi-Atwan M, Bushman J, Bushmann J, Tornovsky-Babaey S, Perry A, Abu-Libdeh A, Glaser B, Shyng SL, Zangen DH. Novel de novo mutation in sulfonylurea receptor 1 presenting as hyperinsulinism in infancy followed by overt diabetes in early adolescence. Diabetes. 57: 1935-40. PMID 18390792 DOI: 10.2337/db08-0159 |
0.625 |
|
2008 |
Lin YW, Bushman JD, Yan FF, Haidar S, MacMullen C, Ganguly A, Stanley CA, Shyng SL. Destabilization of ATP-sensitive potassium channel activity by novel KCNJ11 mutations identified in congenital hyperinsulinism. The Journal of Biological Chemistry. 283: 9146-56. PMID 18250167 DOI: 10.1074/jbc.M708798200 |
0.743 |
|
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