We are testing a new system for linking grants to scientists.
The funding information displayed below comes from the
NIH Research Portfolio Online Reporting Tools and the
NSF Award Database.
The grant data on this page is limited to grants awarded in the United States and is thus partial. It can nonetheless be used to understand how funding patterns influence mentorship networks and vice-versa, which has deep implications on how research is done.
You can help! If you notice any innacuracies, please
sign in and mark grants as correct or incorrect matches.
Sign in to see low-probability grants and correct any errors in linkage between grants and researchers.
High-probability grants
According to our matching algorithm, Youn-Young K. Hong is the likely recipient of the following grants.
| Years |
Recipients |
Code |
Title / Keywords |
Matching
score |
| 2007 — 2009 |
Hong, Youn-Young Kate |
F31Activity Code Description:
To provide predoctoral individuals with supervised research training in specified health and health-related areas leading toward the research degree (e.g., Ph.D.). |
The Role of Sidekicks in Synaptic Specificity
[unreadable] DESCRIPTION (provided by applicant): My broad goal is to elucidate mechanisms by which neurons form specific synapses at precisely targeted locations. One major determinant of synaptic specificity is laminar specificity, the restriction of particular axonal arbors to specific laminae within the target area. The widespread and prominent presence of laminar specificity throughout the central nervous system is indicative of its fundamental importance. Recent work from the Sanes laboratory provided clues that one such family of molecules, Sidekicks (Sdks), mediate this developmental process in the retina. Using the mouse retina as a model system, I will use cellular and genetic approaches to functionally test the role of Sdks. I hypothesize that Sidekicks are determinants of lamina- specific synaptogenesis. [unreadable] [unreadable]
|
1 |
| 2013 — 2014 |
Hong, Youn-Young Kate |
F32Activity Code Description:
To provide postdoctoral research training to individuals to broaden their scientific background and extend their potential for research in specified health-related areas. |
Alternate Pathways For the Spread of Excitation:Functional and Behavioral Studie @ Columbia University Health Sciences
DESCRIPTION (provided by applicant): An essential step in understanding sensory processing is to identify the synaptic circuitry underlying its neural representation. Aberrant network activity that disrupts excitation-inhibition balance can have severe consequences, contributing to the development of epilepsy, schizophrenia or Parkinson's disease. Identifying the mechanisms for the spread of excitation will directly inform about potential abnormalities that arise under pathological conditions. Thalamic projections to cortical layer 4 (L4) are the primary pathway by which sensory information enters the cortex via the thalamus. Information is thought to flow along a hierarchical series of connections from L4 to superficial L2/3, to the output cells in L5/6. However, thalamic neurons send sparse projections to virtually all layers of cortex, notably to the L5/L6 border. Mechanisms may exist that render sparse projections, often ignored in the study of circuits, highly effective in spreading excitatory activity. This would suggest a more complex mechanism of sensory information processing than previously thought, by which information relayed by thalamus reaches cortical output neurons via direct parallel pathways. Using optogenetics, electrophysiology, and behavioral paradigms, we propose to demonstrate that direct thalamocortical inputs can strongly, and directly drive L5 output neurons without L4 activity, and determine how the direct and indirect pathways affect sensory behavior.
|
0.957 |