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High-probability grants
According to our matching algorithm, Ernesto Salcedo is the likely recipient of the following grants.
| Years |
Recipients |
Code |
Title / Keywords |
Matching
score |
| 2003 — 2004 |
Salcedo, Ernesto |
F32Activity Code Description:
To provide postdoctoral research training to individuals to broaden their scientific background and extend their potential for research in specified health-related areas. |
Axonal Targeting in the Mouse Olfactory Bulb @ University of Colorado Denver
DESCRIPTION (provided by applicant): The mechanisms that enable olfactory sensory neurons in the mouse nasal epithelium to target distinct glomeruli in the main olfactory bulb remain unresolved. In order to provide new information on this issue, we propose the following experiments: In Specific Aim 1, we will test the role that major histocompatibility class I molecules play in axon guidance. To accomplish this, we will examine gene-targeted mice that have tau-lacZ labeled olfactory sensory neurons and are deficient in the presentation of class I MHC signaling molecules on cell surfaces. In Specific Aim 2, we will use microarray gene chip technology to identify potential guidance cues that are differentially expressed in the main olfactory bulb. We will also examine the effect that nerve transection has on gene expression. These experiments should significantly further our understanding of axonal targeting in the main olfactory bulb.
|
0.982 |
| 2007 — 2009 |
Salcedo, Ernesto |
R03Activity Code Description:
To provide research support specifically limited in time and amount for studies in categorical program areas. Small grants provide flexibility for initiating studies which are generally for preliminary short-term projects and are non-renewable. |
Major Histocompatibility Class I Molecules in the Main Olfactory System @ University of Colorado Denver
[unreadable] DESCRIPTION (provided by applicant): The mechanisms by which the human brain assembles and maintains a network of over a billion neurons remains a fundamental question in neuroscience. The mouse olfactory system, with its accessible and relatively large olfactory tissue and pool of constantly overturning sensory neurons (OSNs) offers a unique model to examine this question. Our long time goal is to understand the mechanisms involved in the establishing and maintaining the neuronal connections between the main olfactory epithelium and the main olfactory bulb. Recent findings have linked major histocompatibility type I (MHCI) molecules and the MHCI signaling pathway to neuronal restructuring and refinement. We hypothesize that MHCI molecules play a role in OSN axonal guidance to the MOB. This hypothesis is based on our preliminary findings that (1) MHCI molecules can be detected in the glomerular layer of the main olfactory bulb, and (2) there are OSN targeting and glomerular anomalies in MHCI-deficient mice. Our specific aims are: (1) Characterize and localize the expression pattern of MHCI molecules in the mouse olfactory system. (2) Correlate a deficiency in cell surface class I MHC expression with an increase in OSN mistargeting and an alteration of the organization of the glomerular layer of the MOB. [unreadable] [unreadable] [unreadable]
|
0.982 |