Isil Aksan Kurnaz
Affiliations: | 2008-2012 | Genetics and Bioengineering | Yeditepe University, Istanbul Turkey |
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"Isil Kurnaz"Mean distance: 16.28 (cluster 11)
Children
Sign in to add trainee| Oya Ari | grad student | Yeditepe University, Istanbul Turkey | |
| Elif Kon | grad student | Université catholique de Louvain | |
| Melis Savasan Sogut | grad student | 2008- | Yeditepe University, Istanbul Turkey |
| Basak Kandemir | grad student | 2011- | Yeditepe University, Istanbul Turkey |
| Ekin Sonmez | grad student | 2017- | Gebze Technical University |
| Burcu Erdogan | grad student | 2008-2012 | Yeditepe University, Istanbul Turkey |
| Eray Sahin | grad student | 2013-2016 | Yeditepe University, Istanbul Turkey |
| Ugur Dag | research scientist | 2009-2012 |
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Publications
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| Sogut MS, Venugopal C, Kandemir B, et al. (2021) ETS-Domain Transcription Factor Elk-1 Regulates Stemness Genes in Brain Tumors and CD133+ BrainTumor-Initiating Cells. Journal of Personalized Medicine. 11 |
| Babal YK, Kandemir B, Kurnaz IA. (2021) Gene Regulatory Network of ETS Domain Transcription Factors in Different Stages of Glioma. Journal of Personalized Medicine. 11 |
| Kandemir B, Gulfidan G, Arga KY, et al. (2020) Transcriptomic profile of Pea3 family members reveal regulatory codes for axon outgrowth and neuronal connection specificity. Scientific Reports. 10: 18162 |
| Kandemir B, Davis S, Yiğit EN, et al. (2020) Expression of Pea3 protein subfamily members in hippocampus and potential regulation following neuronal stimulation. Neuroscience Letters. 135348 |
| Süer C, Liman N, Kandemir B, et al. (2015) P.1.e.017 A decrease in phosphorylation of CREB may explain delayed place learning and attenuated long-term potentiation in adult-onset hyperthyroidism European Neuropsychopharmacology. 25: S218-S219 |
| Kandemir B, Caglayan B, Hausott B, et al. (2014) Pea3 transcription factor promotes neurite outgrowth. Frontiers in Molecular Neuroscience. 7: 59 |
| Demir O, Kurnaz IA. (2013) Phospho-Ser383-Elk-1 is localized to the mitotic spindles during cell cycle and interacts with mitotic kinase Aurora-A. Cell Biochemistry and Function. 31: 591-8 |
| Mut M, Lule S, Demir O, et al. (2012) Both mitogen-activated protein kinase (MAPK)/extracellular-signal-regulated kinases (ERK) 1/2 and phosphatidylinositide-3-OH kinase (PI3K)/Akt pathways regulate activation of E-twenty-six (ETS)-like transcription factor 1 (Elk-1) in U138 glioblastoma cells. The International Journal of Biochemistry & Cell Biology. 44: 302-10 |
| Demir O, Ari O, Kurnaz IA. (2012) Elk-1 interacts with dynein upon serum stimulation but independent of serine 383 phosphorylation. Cellular and Molecular Neurobiology. 32: 185-9 |
| Demir O, Aysit N, Onder Z, et al. (2011) ETS-domain transcription factor Elk-1 mediates neuronal survival: SMN as a potential target. Biochimica Et Biophysica Acta. 1812: 652-62 |