Richard J. Siarey
Affiliations: | Uniformed Services University of the Health Sciences, Bethesda, MD |
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| Cramer NP, Best TK, Stoffel M, et al. (2010) GABAB-GIRK2-mediated signaling in Down syndrome. Advances in Pharmacology (San Diego, Calif.). 58: 397-426 |
| Baek KH, Zaslavsky A, Lynch RC, et al. (2009) Down's syndrome suppression of tumour growth and the role of the calcineurin inhibitor DSCR1. Nature. 459: 1126-30 |
| Best TK, Cho-Clark M, Siarey RJ, et al. (2008) Speeding of miniature excitatory post-synaptic currents in Ts65Dn cultured hippocampal neurons. Neuroscience Letters. 438: 356-61 |
| Olson LE, Roper RJ, Sengstaken CL, et al. (2007) Trisomy for the Down syndrome 'critical region' is necessary but not sufficient for brain phenotypes of trisomic mice. Human Molecular Genetics. 16: 774-82 |
| Best TK, Siarey RJ, Galdzicki Z. (2007) Ts65Dn, a mouse model of Down syndrome, exhibits increased GABAB-induced potassium current. Journal of Neurophysiology. 97: 892-900 |
| Siarey RJ, Kline-Burgess A, Cho M, et al. (2006) Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome. Journal of Neurochemistry. 98: 1266-77 |
| Harashima C, Jacobowitz DM, Stoffel M, et al. (2006) Elevated expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in cerebellar unipolar brush cells of a Down syndrome mouse model. Cellular and Molecular Neurobiology. 26: 719-34 |
| Harashima C, Jacobowitz DM, Witta J, et al. (2006) Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: a model of Down syndrome. The Journal of Comparative Neurology. 494: 815-33 |
| Siarey RJ, Villar AJ, Epstein CJ, et al. (2005) Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome. Neuropharmacology. 49: 122-8 |
| Galdzicki Z, Siarey RJ. (2003) Understanding mental retardation in Down's syndrome using trisomy 16 mouse models. Genes, Brain, and Behavior. 2: 167-78 |