Amit Agarwal, Ph.D.

Affiliations: 
2004-2010 Neurogenetics Max-Planck-Institute for Experimental Medicine, Göttingen, Germany 
 2010-2017 Neuroscience Johns Hopkins University School of Medicine, Baltimore, MD, United States 
 2018- Institute for Anatomy and Cell Biology Heidelberg Universität 
Area:
Oligodendrocytes, Astrocytes, Glia, Myelin, Mouse Genetics, Physiology, Calcium signaling, Microscopy
Website:
http://www.ana.uni-heidelberg.de/index.php?id=236
Google:
"https://scholar.google.com/citations?user=u0oEE8gAAAAJ&hl=en&oi=ao"
Mean distance: 14.18 (cluster 11)
 
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Publications

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Khawaja RR, Agarwal A, Fukaya M, et al. (2021) GluA2 overexpression in oligodendrocyte progenitors promotes postinjury oligodendrocyte regeneration. Cell Reports. 35: 109147
Escartin C, Galea E, Lakatos A, et al. (2021) Reactive astrocyte nomenclature, definitions, and future directions. Nature Neuroscience
Ye L, Orynbayev M, Zhu X, et al. (2020) Ethanol abolishes vigilance-dependent astroglia network activation in mice by inhibiting norepinephrine release. Nature Communications. 11: 6157
Yoo SW, Agarwal A, Smith MD, et al. (2020) Inhibition of neutral sphingomyelinase 2 promotes remyelination. Science Advances. 6
Venkatesh HS, Morishita W, Geraghty AC, et al. (2019) Electrical and synaptic integration of glioma into neural circuits. Nature
Venkataramani V, Tanev DI, Strahle C, et al. (2019) Glutamatergic synaptic input to glioma cells drives brain tumour progression. Nature
Larson VA, Mironova Y, Vanderpool KG, et al. (2018) Oligodendrocytes control potassium accumulation in white matter and seizure susceptibility. Elife. 7
Larson VA, Mironova Y, Vanderpool KG, et al. (2018) Author response: Oligodendrocytes control potassium accumulation in white matter and seizure susceptibility Elife
Agarwal A, Wu PH, Hughes EG, et al. (2017) Transient Opening of the Mitochondrial Permeability Transition Pore Induces Microdomain Calcium Transients in Astrocyte Processes. Neuron
Perea G, Gómez R, Mederos S, et al. (2016) Activity-dependent switch of GABAergic inhibition into glutamatergic excitation in astrocyte-neuron networks. Elife. 5
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