Patricia Shinnick-Gallagher

University of Texas Medical Branch, Galveston, TX 
"Patricia Shinnick-Gallagher"
Mean distance: 14.86 (cluster 6)


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Robert S. Jacobs grad student UC Santa Barbara (agrizz) Delete Edit
Syogoro Nishi post-doc Kurume University School of Medicine (agrizz) Delete Edit


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Alison E. Cole grad student 1977-1981 UTMB (coleal) Delete Edit
N. Brad Keele grad student 1997 UTMB (agrizz) Delete Edit
Bradley W. Schroeder grad student 2006 UTMB (pq) Delete Edit
Balaji Krishnan post-doc 2005- UTMB (krishnanbalaji_27) Delete Edit
Donald G. Rainnie post-doc 1989-1992 Emory (drainni) Delete Edit
BETA: Related publications


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Krishnan B, Scott MT, Pollandt S, et al. (2015) Fear Potentiated Startle Increases Phospholipase D (PLD) Expression/Activity and PLD-Linked Metabotropic Glutamate Receptor Mediated Post-Tetanic Potentiation in Rat Amygdala. Neurobiology of Learning and Memory
Krishnan B, Genzer KM, Pollandt SW, et al. (2011) Dopamine-induced plasticity, phospholipase D (PLD) activity and cocaine-cue behavior depend on PLD-linked metabotropic glutamate receptors in amygdala. Plos One. 6: e25639
Schmidt K, Krishnan B, Xia Y, et al. (2011) Cocaine withdrawal reduces group I mGluR-mediated long-term potentiation via decreased GABAergic transmission in the amygdala. The European Journal of Neuroscience. 34: 177-89
Krishnan B, Centeno M, Pollandt S, et al. (2010) Dopamine receptor mechanisms mediate corticotropin-releasing factor-induced long-term potentiation in the rat amygdala following cocaine withdrawal. The European Journal of Neuroscience. 31: 1027-42
Billa SK, Liu J, Bjorklund NL, et al. (2010) Increased insertion of glutamate receptor 2-lacking alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid (AMPA) receptors at hippocampal synapses upon repeated morphine administration. Molecular Pharmacology. 77: 874-83
Gallagher JP, Orozco-Cabal LF, Liu J, et al. (2008) Synaptic physiology of central CRH system. European Journal of Pharmacology. 583: 215-25
Orozco-Cabal L, Liu J, Pollandt S, et al. (2008) Dopamine and corticotropin-releasing factor synergistically alter basolateral amygdala-to-medial prefrontal cortex synaptic transmission: functional switch after chronic cocaine administration. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 529-42
Fu Y, Pollandt S, Liu J, et al. (2007) Long-term potentiation (LTP) in the central amygdala (CeA) is enhanced after prolonged withdrawal from chronic cocaine and requires CRF1 receptors. Journal of Neurophysiology. 97: 937-41
Pollandt S, Liu J, Orozco-Cabal L, et al. (2006) Cocaine withdrawal enhances long-term potentiation induced by corticotropin-releasing factor at central amygdala glutamatergic synapses via CRF, NMDA receptors and PKA. The European Journal of Neuroscience. 24: 1733-43
Orozco-Cabal L, Pollandt S, Liu J, et al. (2006) Regulation of synaptic transmission by CRF receptors. Reviews in the Neurosciences. 17: 279-307
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