Matthew J. Spindler, Ph.D.

2011 Pharmaceutical Sciences and Pharmacogenomics University of California, San Francisco, San Francisco, CA 
G protein-coupled receptors
"Matthew Spindler"
Mean distance: 16.21 (cluster 11)


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Bruce  R. Conklin grad student 2011 UCSF
 (Signaling Scaffolds in Cardiovascular Development and Disease.)
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Fenix AM, Miyaoka Y, Bertero A, et al. (2021) Gain-of-function cardiomyopathic mutations in RBM20 rewire splicing regulation and re-distribute ribonucleoprotein granules within processing bodies. Nature Communications. 12: 6324
Medina-Cucurella AV, Mizrahi RA, Asensio MA, et al. (2019) Preferential Identification of Agonistic OX40 Antibodies by Using Cell Lysate to Pan Natively Paired, Humanized Mouse-Derived Yeast Surface Display Libraries. Antibodies (Basel, Switzerland). 8
Johnson KR, Nicodemus-Johnson J, Spindler MJ, et al. (2015) Genome-Wide Gene Expression Analysis Shows AKAP13-Mediated PKD1 Signaling Regulates the Transcriptional Response to Cardiac Hypertrophy. Plos One. 10: e0132474
Taglieri DM, Johnson KR, Burmeister BT, et al. (2014) The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy. Journal of Molecular and Cellular Cardiology. 66: 27-40
Kreitzer FR, Salomonis N, Sheehan A, et al. (2013) A robust method to derive functional neural crest cells from human pluripotent stem cells. American Journal of Stem Cells. 2: 119-31
Spindler MJ, Burmeister BT, Huang Y, et al. (2013) AKAP13 Rho-GEF and PKD-binding domain deficient mice develop normally but have an abnormal response to β-adrenergic-induced cardiac hypertrophy. Plos One. 8: e62705
Salomonis N, Schlieve CR, Pereira L, et al. (2010) Alternative splicing regulates mouse embryonic stem cell pluripotency and differentiation. Proceedings of the National Academy of Sciences of the United States of America. 107: 10514-9
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