Matthew J. Spindler, Ph.D.
Affiliations: | 2011 | Pharmaceutical Sciences and Pharmacogenomics | University of California, San Francisco, San Francisco, CA |
Area:
G protein-coupled receptorsGoogle:
"Matthew Spindler"Mean distance: 16.21 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Bruce R. Conklin | grad student | 2011 | UCSF | |
| (Signaling Scaffolds in Cardiovascular Development and Disease.) | ||||
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Publications
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| Fenix AM, Miyaoka Y, Bertero A, et al. (2021) Gain-of-function cardiomyopathic mutations in RBM20 rewire splicing regulation and re-distribute ribonucleoprotein granules within processing bodies. Nature Communications. 12: 6324 |
| Johnson KR, Nicodemus-Johnson J, Spindler MJ, et al. (2015) Genome-Wide Gene Expression Analysis Shows AKAP13-Mediated PKD1 Signaling Regulates the Transcriptional Response to Cardiac Hypertrophy. Plos One. 10: e0132474 |
| Taglieri DM, Johnson KR, Burmeister BT, et al. (2014) The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy. Journal of Molecular and Cellular Cardiology. 66: 27-40 |
| Kreitzer FR, Salomonis N, Sheehan A, et al. (2013) A robust method to derive functional neural crest cells from human pluripotent stem cells. American Journal of Stem Cells. 2: 119-31 |
| Spindler MJ, Burmeister BT, Huang Y, et al. (2013) AKAP13 Rho-GEF and PKD-binding domain deficient mice develop normally but have an abnormal response to β-adrenergic-induced cardiac hypertrophy. Plos One. 8: e62705 |
| Salomonis N, Schlieve CR, Pereira L, et al. (2010) Alternative splicing regulates mouse embryonic stem cell pluripotency and differentiation. Proceedings of the National Academy of Sciences of the United States of America. 107: 10514-9 |