Takafumi Wataya, MD, Kyoto University,PhD, Kyoto University
Affiliations: | Neurosurgery | Harvard University, Cambridge, MA, United States |
Area:
NeuroscienceWebsite:
http://connects.catalyst.harvard.edu/profiles/profile/person/61466Google:
"Takafumi Wataya"Bio:
http://www.linkedin.com/pub/takafumi-wataya/13/b0a/147
Mean distance: 16.91 (cluster 28) | S | N | B | C | P |
Cross-listing: Neuropathology Tree
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Publications
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Perry G, Ávila J, Casadesus G, et al. (2003) La función del estrés oxidativo en la patogénesis de la enfermedad de Alzheimer Revista Chilena De Neuro-PsiquiatríA. 41 |
Perry G, Ávila J, Casadesus G, et al. (2003) The role of oxidative stress in the pathogenesis of Alzheimer's disease | La función del estrés oxidativo en la patogénesis de la enfermedad de Alzheimer Revista Chilena De Neuro-Psiquiatria. 41: 47-52 |
Perry G, Nunomura A, Cash AD, et al. (2002) Reactive oxygen: its sources and significance in Alzheimer disease. Journal of Neural Transmission. Supplementum. 69-75 |
Wataya T, Nunomura A, Smith MA, et al. (2002) High molecular weight neurofilament proteins are physiological substrates of adduction by the lipid peroxidation product hydroxynonenal. The Journal of Biological Chemistry. 277: 4644-8 |
Nunomura A, Perry G, Aliev G, et al. (2001) Oxidative damage is the earliest event in Alzheimer disease. Journal of Neuropathology and Experimental Neurology. 60: 759-67 |
Perry G, Raina AK, Nunomura A, et al. (2000) How important is oxidative damage? Lessons from Alzheimer's disease. Free Radical Biology & Medicine. 28: 831-4 |
Hattori N, Kitagawa K, Higashida T, et al. (1998) CI-ATPase and Na+/K(+)-ATPase activities in Alzheimer's disease brains. Neuroscience Letters. 254: 141-4 |
Perry G, Wataya T, Nunomura A, et al. (1998) NEUROFILAMENT HEAVY SUBUNIT (NFH) IS A SPECIFIC TARGET OF OXIDATIVE DAMAGE IN THE AXON Journal of Neuropathology and Experimental Neurology. 57: 492 |