Jeffrey Robbins

Affiliations: 
Cincinnati Children's Hospital, Cincinnati, OH, United States 
Area:
Cardiovascular BIology
Website:
https://www.cincinnatichildrens.org/bio/r/jeffrey-robbins
Google:
"Jeffrey Robbins"
Bio:

ROBBINS, JEFFREY, THE PREPARATION AND CHARACTERIZATION OF MYOSIN COMPLEMENTARY DNA (1976). Doctoral Dissertations. AAI7714498.
https://opencommons.uconn.edu/dissertations/AAI7714498

Mean distance: 17 (cluster 11)
 

Parents

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Heinz Herrmann grad student 1977 University of Connecticut (Cell Biology Tree)
 (The preparation and characterization of myosin complementary DNA.)
Stuart MacKenzie Heywood grad student 1977 University of Connecticut (Cell Biology Tree)
BETA: Related publications

Publications

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Qi L, Zang H, Wu W, et al. (2020) CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytes. Journal of Molecular and Cellular Cardiology
Xu N, Gulick J, Osinska H, et al. (2020) Ube2v1 Positively Regulates Protein Aggregation by Modulating Ubiquitin Proteasome System Performance Partially through K63 Ubiquitination. Circulation Research
Zheng D, Su Z, Zhang Y, et al. (2019) Calpain-2 promotes MKP-1 expression protecting cardiomyocytes in both in vitro and in vivo mouse models of doxorubicin-induced cardiotoxicity. Archives of Toxicology
Rahmanseresht S, Lee K, Robbins J, et al. (2019) Resolving the Actin Lattice and Identifying the Relative Position of MYBP-C's N-Terminus in Cardiac Muscle using Storm Microscopy Biophysical Journal. 116: 116a
Meng Q, Bhandary B, Bhuiyan MS, et al. (2018) Myofibroblast-Specific TGFβ Receptor II Signaling in the Fibrotic Response to Cardiac Myosin Binding Protein C-Induced Cardiomyopathy. Circulation Research. 123: 1285-1297
Abdullah CS, Alam S, Aishwarya R, et al. (2018) Cardiac Dysfunction in the Sigma 1 Receptor Knockout Mouse Associated With Impaired Mitochondrial Dynamics and Bioenergetics. Journal of the American Heart Association. 7: e009775
Bhandary B, Meng Q, James J, et al. (2018) Cardiac Fibrosis in Proteotoxic Cardiac Disease is Dependent Upon Myofibroblast TGF -β Signaling. Journal of the American Heart Association. 7: e010013
Lowey S, Bretton V, Joel PB, et al. (2018) Hypertrophic cardiomyopathy R403Q mutation in rabbit β-myosin reduces contractile function at the molecular and myofibrillar levels. Proceedings of the National Academy of Sciences of the United States of America
Baker RS, Zafar F, Kimura N, et al. (2018) In Vivo Remodeling of an Extracellular Matrix Cardiac Patch in an Ovine Model. Asaio Journal (American Society For Artificial Internal Organs : 1992)
Alam S, Abdullah CS, Aishwarya R, et al. (2018) Aberrant Mitochondrial Fission Is Maladaptive in Desmin Mutation-Induced Cardiac Proteotoxicity. Journal of the American Heart Association. 7
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