John K. Robinson

Affiliations: 
Psychology Stony Brook University, Stony Brook, NY, United States 
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Publications

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Popescu DL, Van Nostrand WE, Robinson JK. (2020) Longitudinal Cognitive Decline in a Novel Rodent Model of Cerebral Amyloid Angiopathy Type-1. International Journal of Molecular Sciences. 21
Robison LS, Francis N, Popescu DL, et al. (2020) Environmental Enrichment: Disentangling the Influence of Novelty, Social, and Physical Activity on Cerebral Amyloid Angiopathy in a Transgenic Mouse Model. International Journal of Molecular Sciences. 21
Francis N, Robison LS, Popescu DL, et al. (2019) Voluntary Wheel Running Reduces Amyloid-β42 and Rescues Behavior in Aged Tg2576 Mouse Model of Alzheimer's Disease. Journal of Alzheimer's Disease : Jad
Robison LS, Popescu DL, Anderson ME, et al. (2019) Long-term voluntary wheel running does not alter vascular amyloid burden but reduces neuroinflammation in the Tg-SwDI mouse model of cerebral amyloid angiopathy. Journal of Neuroinflammation. 16: 144
Davis J, Xu F, Hatfield J, et al. (2018) A Novel Transgenic Rat Model of Robust Cerebral Microvascular Amyloid with Prominent Vasculopathy. The American Journal of Pathology
Caponegro MD, Torres LF, Rastegar C, et al. (2018) Pifithrin-μ modulates microglial activation and promotes histological recovery following spinal cord injury. Cns Neuroscience & Therapeutics
Robison LS, Popescu DL, Anderson ME, et al. (2018) The effects of volume versus intensity of long-term voluntary exercise on physiology and behavior in C57/Bl6 mice. Physiology & Behavior
Anderson M, Xu F, Ou-Yang MH, et al. (2016) Intensive 'Brain Training' Intervention Fails to Reduce Amyloid Pathologies or Cognitive Deficits in Transgenic Mouse Models of Alzheimer's Disease. Journal of Alzheimer's Disease : Jad
Vasek MJ, Garber C, Dorsey D, et al. (2016) A complement-microglial axis drives synapse loss during virus-induced memory impairment. Nature. 534: 538-543
Birey F, Kloc M, Chavali M, et al. (2015) Genetic and Stress-Induced Loss of NG2 Glia Triggers Emergence of Depressive-like Behaviors through Reduced Secretion of FGF2. Neuron
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