Gregorio Valdez, Ph.D.
Affiliations: | 2005 | Stony Brook University, Stony Brook, NY, United States |
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Parents
Sign in to add mentorSimon Halegoua | grad student | 2005 | SUNY Stony Brook | |
(Macroendocytosis underlies retrograde axonal signaling of Trk receptors.) | ||||
Joshua R. Sanes | post-doc | Harvard | ||
Eleanore T. Wurtzel | research scientist | Lehman College, CUNY (Chemistry Tree) |
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Publications
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Juros D, Avila MF, Hastings RL, et al. (2024) Cellular and molecular alterations to muscles and neuromuscular synapses in a mouse model of MEGF10-related myopathy. Skeletal Muscle. 14: 10 |
Hastings RL, Valdez G. (2024) Origin, identity, and function of terminal Schwann cells. Trends in Neurosciences |
Hastings RL, Avila MF, Suneby E, et al. (2023) Cellular and molecular evidence that synaptic Schwann cells contribute to aging of mouse neuromuscular junctions. Aging Cell. 22: e13981 |
Procacci NM, Hastings RL, Aziz AA, et al. (2022) Kir4.1 is specifically expressed and active in non-myelinating Schwann cells. Glia |
Larouche J, Mohiuddin M, Choi JJ, et al. (2021) Murine muscle stem cell response to perturbations of the neuromuscular junction are attenuated with aging. Elife. 10 |
Valdez G. (2019) Effects of disease-afflicted and aging neurons on the musculoskeletal system. Bone. 122: 31-37 |
Taetzsch T, Valdez G. (2018) NMJ maintenance and repair in aging. Current Opinion in Physiology. 4: 57-64 |
Vaughan SK, Sutherland NM, Zhang S, et al. (2018) The ALS-inducing factors, TDP43 and SOD1, directly affect and sensitize sensory neurons to stress. Scientific Reports. 8: 16582 |
Herskovits AZ, Hunter TA, Maxwell N, et al. (2018) SIRT1 deacetylase in aging-induced neuromuscular degeneration and amyotrophic lateral sclerosis. Aging Cell. e12839 |
Magalhães-Gomes MPS, Motta-Santos D, Schetino LPL, et al. (2018) Fast and slow-twitching muscles are differentially affected by reduced cholinergic transmission in mice deficient for VAChT: A mouse model for congenital myasthenia. Neurochemistry International |