Monica Driscoll

Affiliations: 
Rutgers University, New Brunswick, New Brunswick, NJ, United States 
Area:
C. elegans, neurodegeneration, neuroregeneration
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"Monica Driscoll"
Mean distance: 15.61 (cluster 11)
 
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Publications

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Yang Y, Arnold ML, Lange CM, et al. (2024) Autophagy protein ATG-16.2 and its WD40 domain mediate the beneficial effects of inhibiting early-acting autophagy genes in C. elegans neurons. Nature Aging
Wang G, Guasp R, Salam S, et al. (2023) Mechanical force of uterine occupation enables large vesicle extrusion from proteostressed maternal neurons. Biorxiv : the Preprint Server For Biology
Arnold ML, Cooper J, Androwski R, et al. (2023) Intermediate filaments associate with aggresome-like structures in proteostressed C. elegans neurons and influence large vesicle extrusions as exophers. Nature Communications. 14: 4450
Wang Y, Arnold ML, Smart AJ, et al. (2023) Large vesicle extrusions from neurons are consumed and stimulated by glial-like phagocytosis activity of the neighboring cell. Elife. 12
Cooper JF, Guasp RJ, Arnold ML, et al. (2021) Stress increases in exopher-mediated neuronal extrusion require lipid biosynthesis, FGF, and EGF RAS/MAPK signaling. Proceedings of the National Academy of Sciences of the United States of America. 118
Arnold ML, Cooper J, Grant BD, et al. (2020) Quantitative Approaches for Scoring in vivo Neuronal Aggregate and Organelle Extrusion in Large Exopher Vesicles in C. elegans. Journal of Visualized Experiments : Jove
Chen Y, Onken B, Chen H, et al. (2020) Healthy lifespan extension mediated by oenothein B isolated from Eucalyptus grandis × Eucalyptus urophylla GL9 in Caenorhabditis elegans. Food & Function
Driscoll M. (2020) Neurons Putting Out the Trash: A Novel Facet of Proteostasis and Mitochondrial Quality Control Innovation in Aging. 4: 738-738
Lesanpezeshki L, Hewitt JE, Laranjeiro R, et al. (2019) Pluronic gel-based burrowing assay for rapid assessment of neuromuscular health in C. elegans. Scientific Reports. 9: 15246
Wang G, Sun L, Reina CP, et al. (2019) CED-4 CARD domain residues can modulate non-apoptotic neuronal regeneration functions independently from apoptosis. Scientific Reports. 9: 13315
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