Steven W. Barger

Affiliations: 
University of Arkansas for Medical Sciences, United States 
Area:
Alzheimer's disease, neuroinflammation, microglia, gene regulation, glutamatergic neurotransmission
Website:
http://www.uams.edu/neuroscience_cellbiology/faculty/details.asp?id=62
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"Steven Barger"
Bio:

B.A. (Biology) Hendrix College, 1987
Ph.D. (Cell biology) Vanderbilt U., 1992
Postdoc (w/ Mark Mattson) Sanders-Brown Ctr. on Aging, U. Kentucky, 1995
Asst. Prof. --> Professor (Geriatrics; Neurobiology & Developmental Sciences) UAMS, 1995-present

Mean distance: 16.56 (cluster 41)
 
SNBCP
Cross-listing: Alzheimer's Tree

Children

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Xianrong Mao grad student 1997-2001 UAMS
Shengzhou Wu grad student 2004 UAMS
Wei Wang grad student 2005-2010 UAMS
Rachel D. Hendrix grad student 2012-2018 UAMS
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Publications

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Parcon PA, Balasubramaniam M, Ayyadevara S, et al. (2017) Apolipoprotein E4 inhibits autophagy gene products through direct, specific binding to CLEAR motifs. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
Wang W, Moerman-Herzog AM, Slaton A, et al. (2016) Presenilin 1 mutations influence processing and trafficking of the ApoE receptor apoER2. Neurobiology of Aging. 49: 145-153
Mehdi SJ, Rosas-Hernandez H, Cuevas E, et al. (2016) Protein Kinases and Parkinson's Disease. International Journal of Molecular Sciences. 17
Wu S, Barger SW. (2016) Disrupted-in-schizophrenia-1 as a broader link of glutamatergic transmission to schizophrenia impacts cerebral neurochemistry via the production of the 'gliotransmitter' d-serine, a NMDA receptor coagonist. Journal of Neurochemistry. 138: 503-505
Ayyadevara S, Balasubramaniam M, Parcon PA, et al. (2016) Proteins that mediate protein aggregation and cytotoxicity distinguish Alzheimer's hippocampus from normal controls. Aging Cell
Mao X, Phanavanh B, Hamdan H, et al. (2016) NFκB-inducing kinase inhibits NFκB activity specifically in neurons of the CNS. Journal of Neurochemistry
Deng J, Habib A, Obregon DF, et al. (2015) Soluble amyloid precursor protein alpha (sAPPα) inhibits tau phosphorylation through modulation of GSK3β signaling pathway. Journal of Neurochemistry
Kuang XL, Liu F, Chen H, et al. (2014) Reductions of the components of the calreticulin/calnexin quality-control system by proteasome inhibitors and their relevance in a rodent model of Parkinson's disease. Journal of Neuroscience Research. 92: 1319-29
Barger SW. (2013) TMEM106B and frontotemporal lobar degeneration: can over-expression tell us how reductions are beneficial? Journal of Neurochemistry. 126: 696-8
Bailey AR, Hou H, Song M, et al. (2013) GFAP expression and social deficits in transgenic mice overexpressing human sAPPα. Glia. 61: 1556-69
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