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Timothy M. Barth

Affiliations: 
Texas Christian University, Fort Worth, TX, United States 
Area:
plasticity, recovery of function
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"Timothy Barth"
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Parents

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Timothy Schallert grad student UT Austin
Steven P. Wise post-doc NIMH

Children

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Michael R. Hoane grad student Texas Christian University
Troy Schmanke grad student 1990-1994 Texas Christian University
Jocelyn J. Cooledge grad student 2002 Texas Christian University
Kevin Heinrich grad student 2003 Texas Christian University (Computational Biology Tree)
Jennifer A. Konen grad student 2003 Texas Christian University
Jason F. Mishalanie grad student 2003 Texas Christian University
Pamela M. Stuntz grad student 2003 Texas Christian University
Stefanie L. Byler grad student 2007 Texas Christian University
Kristina L. Dandy grad student 2009 Texas Christian University
Crystal M. Kreitler grad student 2011 Texas Christian University
Julie S. Gray grad student 2012 Texas Christian University

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Publications

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Kreitler CM, Dansereau DF, Barth TM, et al. (2012) Evaluation of a Cognitive Tool for Enhanced Decision Making and Personal Change among College Students Nacada Journal. 32: 18-29
Byler SL, Boehm GW, Karp JD, et al. (2009) Systemic lipopolysaccharide plus MPTP as a model of dopamine loss and gait instability in C57Bl/6J mice. Behavioural Brain Research. 198: 434-9
Byler SL, Shaffer MC, Barth TM. (2006) Unilateral pallidotomy produces motor deficits and excesses in rats. Restorative Neurology and Neuroscience. 24: 133-45
Hoane MR, Barth TM. (2002) The window of opportunity for administration of magnesium therapy following focal brain injury is 24 h but is task dependent in the rat. Physiology & Behavior. 76: 271-80
Hoane MR, Barth TM. (2001) The behavioral and anatomical effects of MgCl2 therapy in an electrolytic lesion model of cortical injury in the rat. Magnesium Research : Official Organ of the International Society For the Development of Research On Magnesium. 14: 51-63
Hoane MR, Barbay S, Barth TM. (2000) Large cortical lesions produce enduring forelimb placing deficits in un-treated rats and treatment with NMDA antagonists or anti-oxidant drugs induces behavioral recovery. Brain Research Bulletin. 53: 175-86
Hoane MR, Irish SL, Marks BB, et al. (1998) Preoperative regimens of magnesium facilitate recovery of function and prevent subcortical atrophy following lesions of the rat sensorimotor cortex. Brain Research Bulletin. 45: 45-51
Hoane MR, Raad C, Barth TM. (1997) Non-competitive NMDA antagonists and anti-oxidant drugs reduce striatal atrophy and facilitate recovery of function following lesions of the rat cortex. Restorative Neurology and Neuroscience. 11: 71-82
Schmanke T, Barth TM. (1997) Amphetamine and task-specific practice augment recovery of vibrissae-evoked forelimb placing after unilateral sensorimotor cortical injury in the rat. Journal of Neurotrauma. 14: 459-68
Schmanke TD, Avery RA, Barth TM. (1996) The effects of amphetamine on recovery of function after cortical damage in the rat depend on the behavioral requirements of the task. Journal of Neurotrauma. 13: 293-307
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